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Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms
被引:0
|作者:
Antonio Rodríguez-Blázquez
Arturo Carabias
Alba Morán-Vaquero
Sergio de Cima
Juan R. Luque-Ortega
Carlos Alfonso
Peter Schuck
José Antonio Manso
Sandra Macedo-Ribeiro
Carmen Guerrero
José M. de Pereda
机构:
[1] Universidad de Salamanca,Centro de Investigación del Cáncer and Instituto de Biología Molecular y Celular del Cáncer, Consejo Superior de Investigaciones Científicas (CSIC)
[2] Instituto de Investigación Biomédica de Salamanca (IBSAL),Laboratory of Dynamics of Macromolecular Assembly, National Institute of Biomedical Imaging and Bioengineering
[3] Centro de Investigaciones Biológicas Margarita Salas,IBMC
[4] CSIC,Instituto de Biologia Molecular e Celular
[5] National Institutes of Health,i3S
[6] Universidade do Porto,Instituto de Investigação e Inovação em Saúde
[7] Universidade do Porto,Departamento de Medicina
[8] Universidad de Salamanca,Structural Molecular Biology Group, Novo Nordisk Foundation Centre for Protein Research, Faculty of Health and Medical Sciences
[9] University of Copenhagen,undefined
来源:
关键词:
Ras-associated protein 1;
RapGEF1;
Signal transduction;
Tyrosine phosphorylation;
Src-homology 2 domain;
Src-homology 3 domain;
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