Microtubule-Tau Interaction as a Therapeutic Target for Alzheimer's Disease

被引:11
|
作者
Pachima, Yanina Ivashko [1 ,2 ]
Zhou, Liu-yao [3 ,4 ]
Lei, Peng [3 ,4 ,5 ]
Gozes, Illana [1 ,2 ]
机构
[1] Tel Aviv Univ, Lily & Avraham Gildor Chair Invest Growth Factors, Dept Human Mol Genet & Biochem,Adams Super Ctr Br, Elton Lab Neuroendocrinol,Sackler Fac Med, IL-69978 Tel Aviv, Israel
[2] Tel Aviv Univ, Sagol Sch Neurosci, IL-69978 Tel Aviv, Israel
[3] Sichuan Univ, West China Hosp, State Key Lab Biotherapy, Dept Neurol, Chengdu 610041, Sichuan, Peoples R China
[4] Collaborat Innovat Ctr Biotherapy, Chengdu 610041, Sichuan, Peoples R China
[5] Univ Melbourne, Florey Inst Neurosci & Mental Hlth, Oxidat Biol Unit, Parkville, Vic 3052, Australia
基金
中国国家自然科学基金; 以色列科学基金会;
关键词
DEPENDENT NEUROPROTECTIVE PROTEIN; AMYLOID-BETA-PROTEIN; PAIRED HELICAL FILAMENT; SIGNAL-REGULATED KINASE; FAST AXONAL-TRANSPORT; NAP DAVUNETIDE; MOUSE MODEL; NEURONAL DIFFERENTIATION; ENDOGENOUS TAU; DRUG CANDIDATE;
D O I
10.1007/s12031-016-0715-x
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
引用
收藏
页码:145 / 152
页数:8
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