Defective axonal transport increases Aβ production downstream from kinesin-1-assisted phosphorylation of amyloid-β precursor protein by JNK

被引：0

|

作者：

Muresan, V. ^{[1
]}

Muresan, Z. Ladescu ^{[1
]}

机构：

[1] Rutgers State Univ, New Jersey Med Sch, Pharmacol & Physiol, Newark, NJ 07102 USA

来源：

MOLECULAR BIOLOGY OF THE CELL | 2014年 / 25卷

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D O I：

暂无

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

P951

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页数：2

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[41] THE MUSCARINIC M1 AGONIST XANOMELINE INCREASES SOLUBLE AMYLOID PRECURSOR PROTEIN RELEASE FROM CHINESE-HAMSTER OVARY-M1 CELLS
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BYMASTER, FP
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[42] The Schizophrenia-Related Protein, DISC1, Controls Production of Amyloid-β (Aβ), a Pathogenic Peptide Implicated in Alzheimer's Disease (AD), by Regulating Trafficking of Aβ Precursor Protein (APP) along Secretory, Endocytic, and Degradative Routes.
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