SENSITIZATION OF THE HOST IMMUNE-SYSTEM TO CENTRAL-NERVOUS-SYSTEM ANTIGENS FOLLOWING EXPERIMENTAL SPINAL-CORD INJURY - A MODEL FOR TRAUMATICALLY INDUCED AUTOIMMUNE-DISEASE

被引：0

作者：

POPOVICH, PG

STOKES, BT

WHITACRE, CC

机构：

[1] OHIO STATE UNIV,COLL MED,DEPT PHYSIOL,COLUMBUS,OH 43210

[2] OHIO STATE UNIV,COLL MED,DEPT MED MICROBIOL & IMMUNOL,COLUMBUS,OH 43210

来源：

FASEB JOURNAL | 1994年 / 8卷 / 04期

关键词：

D O I：

暂无

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：A247 / A247

页数：1

共 45 条

[31] DISTRIBUTION OF NERVE GROWTH-FACTOR RECEPTOR-LIKE IMMUNOREACTIVITY IN THE ADULT-RAT CENTRAL-NERVOUS-SYSTEM - EFFECT OF COLCHICINE AND CORRELATION WITH THE CHOLINERGIC SYSTEM .2. BRAIN-STEM, CEREBELLUM AND SPINAL-CORD
PIORO, EP
CUELLO, AC
NEUROSCIENCE, 1990, 34 (01) : 89 - 110
[32] MODEL FOR THE STUDY OF PLASTICITY OF THE HUMAN NERVOUS-SYSTEM - FEATURES OF RESIDUAL SPINAL-CORD MOTOR-ACTIVITY RESULTING FROM ESTABLISHED POST-TRAUMATIC INJURY
DIMITRIJEVIC, MR
CIBA FOUNDATION SYMPOSIA, 1988, 138 : 227 - 239
[33] CENTRAL NERVOUS-SYSTEM DEMYELINATION IN VENEZUELAN EQUINE ENCEPHALOMYELITIS INFECTION - AN EXPERIMENTAL-MODEL OF VIRUS-INDUCED MYELIN INJURY
DALCANTO, MC
RABINOWITZ, SG
JOURNAL OF THE NEUROLOGICAL SCIENCES, 1981, 49 (03) : 397 - 418
[34] CONDUCTION ABNORMALITIES ARE RESTRICTED TO THE CENTRAL-NERVOUS-SYSTEM IN EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS INDUCED BY INOCULATION WITH PROTEOLIPID PROTEIN BUT NOT WITH MYELIN BASIC-PROTEIN (VOL 117, PG 975, 1994)
CHALK, JB
MCCOMBE, PA
PENDER, MP
BRAIN, 1995, 118 : 1073 - 1073
[35] Central nervous system infiltrates are characterized by features of ongoing B cell-related immune activity in MP4-induced experimental autoimmune encephalomyelitis
Batoulis, Helena
Wunsch, Marie
Birkenheier, Johannes
Rottlaender, Andrea
Gorboulev, Valentin
Kuerten, Stefanie
CLINICAL IMMUNOLOGY, 2015, 158 (01) : 47 - 58
[36] GRAFT-VS-HOST DISEASE ELICITS EXPRESSION OF CLASS-I AND CLASS-II HISTOCOMPATIBILITY ANTIGENS AND THE PRESENCE OF SCATTERED LYMPHOCYTES-T IN RAT CENTRAL-NERVOUS-SYSTEM
HICKEY, WF
KIMURA, H
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1987, 84 (07) : 2082 - 2086
[37] FURTHER-STUDIES ON FREE-RADICAL PATHOLOGY IN THE MAJOR CENTRAL NERVOUS-SYSTEM DISORDERS - EFFECT OF VERY HIGH-DOSES OF METHYLPREDNISOLONE ON THE FUNCTIONAL OUTCOME, MORPHOLOGY, AND CHEMISTRY OF EXPERIMENTAL SPINAL-CORD IMPACT INJURY
DEMOPOULOS, HB
FLAMM, ES
SELIGMAN, ML
PIETRONIGRO, DD
TOMASULA, J
DECRESCITO, V
CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY, 1982, 60 (11) : 1415 - 1424
[38] Tumor necrosis factor blockade in actively induced experimental autoimmune encephalomyelitis prevents clinical disease despite activated T cell infiltration to the central nervous system
Korner, H
Lemckert, FA
Chaudhri, G
Etteldorf, S
Sedgwick, JD
EUROPEAN JOURNAL OF IMMUNOLOGY, 1997, 27 (08) : 1973 - 1981
[39] CLASS-II-RESTRICTED T-CELL RESPONSES IN THEILERS MURINE ENCEPHALOMYELITIS VIRUS (TMEV)-INDUCED DEMYELINATING DISEASE .2. SURVEY OF HOST IMMUNE-RESPONSES AND CENTRAL-NERVOUS-SYSTEM VIRUS TITERS IN INBRED MOUSE STRAINS
CLATCH, RJ
LIPTON, HL
MILLER, SD
MICROBIAL PATHOGENESIS, 1987, 3 (05) : 327 - 337
[40] APOPTOTIC ELIMINATION OF V-BETA-8.2(+) CELLS FROM THE CENTRAL-NERVOUS-SYSTEM DURING RECOVERY FROM EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS INDUCED BY THE PASSIVE TRANSFER OF V-BETA-8.2(+) ENCEPHALITOGENIC T-CELLS
TABI, Z
MCCOMBE, PA
PENDER, MP
EUROPEAN JOURNAL OF IMMUNOLOGY, 1994, 24 (11) : 2609 - 2617

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