CIS-UROCANIC ACID AND HISTAMINE AUGMENT TNF-ALPHA MEDIATED INDUCTION OF KERATINOCYTE ICAM-1 EXPRESSION AND SUPPRESS IFN-GAMMA INDUCTION OF HLA-DR

被引：0

作者：

MITRA, RS

SHIMIZU, Y

NICKOLOFF, BJ

机构：

[1] UNIV MICHIGAN,DEPT PATHOL,ANN ARBOR,MI 48109

[2] UNIV MICHIGAN,DEPT MICROBIOL IMMUNOL,ANN ARBOR,MI 48109

来源：

CLINICAL RESEARCH | 1993年 / 41卷 / 02期

关键词：

D O I：

暂无

中图分类号：

R-3 [医学研究方法]; R3 [基础医学];

学科分类号：

1001 ;

摘要：

引用

页码：A470 / A470

页数：1

共 44 条

[31] MUTUAL ANTAGONISM BETWEEN INTERFERON-GAMMA AND TUMOR-NECROSIS-FACTOR-ALPHA ON FIBROBLAST-LIKE SYNOVIOCYTES - PARADOXICAL INDUCTION OF IFN-GAMMA AND TNF-ALPHA RECEPTOR EXPRESSION
ALVAROGRACIA, JM
YU, C
ZVAIFLER, NJ
FIRESTEIN, GS
JOURNAL OF CLINICAL IMMUNOLOGY, 1993, 13 (03) : 212 - 218
[32] The release of tumor necrosis factor alpha (TNF-alpha) by interferon gamma (IFN-gamma) induced THP-1 cells stimulated with smooth lipopolysaccharide is inhibited by MAbs against HLA-DR and CD14 receptors on the effector cell
Raponi, G
Ghezzi, MC
Mancini, C
MICROBIOLOGICA, 1997, 20 (01): : 1 - 6
[33] Keratinocytes from atopic dermatitis patients show normal induction of HLA-DR and ICAM-1, but enhanced cytokine production, in response to interferon-gamma in vitro.
Pastore, S
Corinti, S
Girolomoni, G
JOURNAL OF INVESTIGATIVE DERMATOLOGY, 1997, 108 (04) : 325 - 325
[34] CYTOKINE (IFN-ALPHA, IFN-GAMMA, IL-1-ALPHA, TNF-ALPHA)-INDUCED MODULATION OF HLA CELL-SURFACE EXPRESSION IN HUMAN BREAST-CANCER CELL-LINES
SEDLAK, J
SPEISER, P
ZEILLINGER, R
KRUGLUGER, W
WILTSCHKE, C
KUBISTA, E
CHORVATH, B
NEOPLASMA, 1992, 39 (05) : 269 - 272
[35] SYNERGISTIC ACTIVATION OF INTERCELLULAR-ADHESION MOLECULE-1 (ICAM-1) BY TNF-ALPHA AND IFN-GAMMA IS MEDIATED BY P65/P50 AND P65/1C-REL AND INTERFERON-RESPONSIVE FACTOR STAT1-ALPHA (P91) THAT CAN BE ACTIVATED BY BOTH IFN-GAMMA AND IFN-ALPHA
JAHNKE, A
JOHNSON, JP
FEBS LETTERS, 1994, 354 (02) : 220 - 226
[36] ROLE OF ICAM-1 AND HLA-DR IN ACCESSORY CELL-FUNCTION OF GAMMA-INTERFERON (IFN-GAMMA) TREATED KERATINOCYTES USING LECTIN, SUPERANTIGEN, AND IMMOBILIZED CD3 MAB AS T-CELL MITOGENS
NICKOLOFF, B
MITRA, R
SHIMIZU, Y
THOMPSON, C
TURKA, L
CLINICAL RESEARCH, 1993, 41 (02): : A179 - A179
[37] ROLE OF ICAM-1 AND HLA-DR IN ACCESSORY CELL-FUNCTION OF GAMMA-INTERFERON (IFN-GAMMA) TREATED KERATINOCYTES USING LECTIN, SUPERANTIGEN, AND IMMOBILIZED CD3 MAB AS T-CELL MITOGENS
NICKOLOFF, B
MITRA, R
SHIMIZU, Y
THOMPSON, C
TURKA, L
JOURNAL OF INVESTIGATIVE DERMATOLOGY, 1993, 100 (04) : 522 - 522
[38] TUMOR-DERIVED FACTOR SYNERGIZES WITH IFN-GAMMA AND LPS, IL-2 OR TNF-ALPHA TO PROMOTE MACROPHAGE SYNTHESIS OF TNF-ALPHA AND TNF RECEPTORS FOR AUTOCRINE INDUCTION OF NITRIC-OXIDE SYNTHASE AND ENHANCED NITRIC OXIDE-MEDIATED TUMOR-CYTOTOXICITY
JIANG, H
STEWART, CA
LEU, RW
IMMUNOBIOLOGY, 1995, 192 (05) : 321 - 342
[39] Effect of human cytokines (IFN-gamma, TNF-alpha, IL-1 beta, IL-4) on porcine endothelial cells: Induction of MHC and adhesion molecules and functional significance of these changes
Batten, P
Yacoub, MH
Rose, ML
IMMUNOLOGY, 1996, 87 (01) : 127 - 133
[40] IL-1 AND TUMOR-NECROSIS-FACTOR-ALPHA EACH UP-REGULATE BOTH THE EXPRESSION OF IFN-GAMMA RECEPTORS AND ENHANCE IFN-GAMMA-INDUCED HLA-DR EXPRESSION ON HUMAN MONOCYTES AND A HUMAN MONOCYTIC CELL-LINE (THP-1)
KRAKAUER, T
OPPENHEIM, JJ
JOURNAL OF IMMUNOLOGY, 1993, 150 (04): : 1205 - 1211

← 1 2 3 4 5 →