MAST-CELLS - IMMUNOLOGICALLY SPECIFIC EFFECTORS AND POTENTIAL SOURCES OF MULTIPLE CYTOKINES DURING IGE-DEPENDENT RESPONSES

被引：0

作者：

GALLI, SJ

WERSHIL, BK

GORDON, JR

MARTIN, TR

机构：

[1] HARVARD UNIV, SCH MED, BOSTON, MA 02215 USA

[2] BETH ISRAEL HOSP, CHARLES A DANA RES INST, BOSTON, MA 02215 USA

[3] HARVARD UNIV, SCH MED, COMBINED PEDIAT GASTROENTEROL & NUTR PROGRAM, BOSTON, MA 02115 USA

[4] CHILDRENS HOSP MED CTR, DEPT PEDIAT, INA SUE PERLMUTTER LAB, BOSTON, MA 02115 USA

来源：

CIBA FOUNDATION SYMPOSIA | 1989年 / 147卷

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：53 / 73

页数：21

共 44 条

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[32] ROLE OF MAST-CELLS VERSUS BASOPHILS IN IGE-DEPENDENT LOCAL EAR SKIN RELEASE OF THE SEROTONIN REQUIRED TO INITIATE CONTACT SENSITIVITY IN MICE
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[33] STUDIES ON THE RECEPTOR MEDIATING CYCLIC AMP-INDEPENDENT ENHANCEMENT BY ADENOSINE OF IGE-DEPENDENT MEDIATOR RELEASE FROM RAT MAST-CELLS
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[34] I-125 FIBRIN DEPOSITION IN IGE-DEPENDENT IMMEDIATE HYPERSENSITIVITY REACTIONS IN MOUSE SKIN - DEMONSTRATION OF THE ROLE OF MAST-CELLS USING GENETICALLY MAST CELL-DEFICIENT MICE LOCALLY RECONSTITUTED WITH CULTURED MAST-CELLS
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[35] DEXAMETHASONE (DEX) INHIBITS IGE-DEPENDENT TNF-ALPHA PRODUCTION BY MOUSE MAST-CELLS (MCS) INVITRO AND SUPPRESSES IGE-ALPHA-DEPENDENT MAST CELL-ALPHA-DEPENDENT AND TNF-ALPHA-DEPENDENT INFLAMMATION INVIVO
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[36] ADENOSINE INHIBITS AND POTENTIATES IGE-DEPENDENT HISTAMINE-RELEASE FROM HUMAN-LUNG MAST-CELLS BY AN A2-PURINOCEPTOR MEDIATED MECHANISM
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[37] THE INITIAL IGE-DEPENDENT INCREASE IN DIACYLGLYCEROL (DAG) PRODUCTION IN RBL 2H3 MAST-CELLS IS INDEPENDENT OF TYROSINE KINASE (TK) ACTIVATION
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[38] HUMAN MAST-CELL HETEROGENEITY - HISTAMINE-RELEASE FROM MAST-CELLS DISPERSED FROM SKIN, LUNG, ADENOIDS, TONSILS, AND COLON IN RESPONSE TO IGE-DEPENDENT AND NONIMMUNOLOGIC STIMULI
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[39] MECHANISM OF INHIBITION OF IGE-DEPENDENT HISTAMINE-RELEASE FROM RAT MAST-CELLS BY XESTOBERGSTEROL-A FROM THE OKINAWAN MARINE SPONGE XESTOSPONGIA-BERGQUISTIA
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[40] THE HUMAN RECOMBINANT C-KIT RECEPTOR LIGAND, RHSCF, INDUCES MEDIATOR RELEASE FROM HUMAN CUTANEOUS MAST-CELLS AND ENHANCES IGE-DEPENDENT MEDIATOR RELEASE FROM BOTH SKIN MAST-CELLS AND PERIPHERAL-BLOOD BASOPHILS
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