EVIDENCE THAT THE RAD1 AND RAD10 PROTEINS OF SACCHAROMYCES-CEREVISIAE PARTICIPATE AS A COMPLEX IN NUCLEOTIDE EXCISION-REPAIR OF UV-RADIATION DAMAGE

被引：12

作者：

SIEDE, W ^{[1
]}

FRIEDBERG, AS ^{[1
]}

FRIEDBERG, EC ^{[1
]}

机构：

[1] UNIV TEXAS, SW MED CTR, DEPT PATHOL, MOLEC PATHOL LAB, DALLAS, TX 75235 USA

来源：

JOURNAL OF BACTERIOLOGY | 1993年 / 175卷 / 19期

关键词：

D O I：

10.1128/JB.175.19.6345-6347.1993

中图分类号：

Q93 [微生物学];

学科分类号：

071005 ; 100705 ;

摘要：

A newly characterized rad1 missense mutation (rad1-20) in the yeast Saccharomyces cerevisiae maps to a region of the Rad1 polypeptide known to be required for Rad1-Rad10 complex formation. The UV sensitivity of the rad1-20 mutant can be partially and specifically corrected by overexpression of wild-type Rad10 protein. These results suggest that complex formation between the Rad1 and Rad10 proteins is required for nucleotide excision repair.

引用

页码：6345 / 6347

页数：3

共 48 条

[41] UV-INDUCED MUTABILITY IN REPAIR-DEFICIENT RAD6-1 STRAINS OF SACCHAROMYCES-CEREVISIAE IS CAUSED BY A SUPPRESSOR GENE
VLCKOVA, V
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BROZMANOVA, J
FOLIA MICROBIOLOGICA, 1992, 37 (04) : 267 - 272
[42] RAD9, RAD24, RAD16 and RAD26 are required for the inducible nucleotide excision repair of UV-induced cyclobutane pyrimidine dimers from the transcribed and non-transcribed regions of the Saccharomyces cerevisiae MFA2 gene
Yu, SR
Teng, YM
Lowndes, NF
Waters, R
MUTATION RESEARCH-DNA REPAIR, 2001, 485 (03): : 229 - 236
[43] UV-induced endonuclease III-sensitive sites at the mating type loci in Saccharomyces cerevisiae are repaired by nucleotide excision repair: RAD7 and RAD16 are not required for their removal from HML alpha
Reed, SH
Boiteux, S
Waters, R
MOLECULAR & GENERAL GENETICS, 1996, 250 (04): : 505 - 514
[44] Requirement of Nse1, a subunit of the Smc5-Smc6 complex, for Rad52-dependent postreplication repair of UV-Damaged DNA in Saccharomyces cerevisiae
Maria, Sergio R. Santa
Gangavarapu, Venkateswarlu
Johnson, Robert E.
Prakash, Louise
Prakash, Satya
MOLECULAR AND CELLULAR BIOLOGY, 2007, 27 (23) : 8409 - 8418
[45] EVIDENCE FOR NUCLEUS INDEPENDENT STEPS IN CONTROL OF REPAIR OF MITOCHONDRIAL DAMAGE .1. UV-INDUCTION OF CYTOPLASMIC PETITE MUTATION IN UV-SENSITIVE NUCLEAR MUTANTS OF SACCHAROMYCES-CEREVISIAE
MOUSTACCHI, E
MOLECULAR AND GENERAL GENETICS, 1972, 114 (01): : 50 - +
[46] Abasic sites linked to dUTP incorporation in DNA are a major cause of spontaneous mutations in absence of base excision repair and Rad17-Mec3-Ddc1 (9-1-1) DNA damage checkpoint clamp in Saccharomyces cerevisiae
Collura, Ada
Van der Kemp, Patricia Auffret
Boiteux, Serge
DNA REPAIR, 2012, 11 (03) : 294 - 303
[47] Inducible nucleotide excision repair (NER) of UV-induced cyclobutane pyrimidine dimers in the cell cycle of the budding yeast Saccharomyces cerevisiae: Evidence that inducible NER is confined to the G1 phase of the mitotic cell cycle
Scott, AD
Waters, R
MOLECULAR & GENERAL GENETICS, 1997, 254 (01): : 43 - 53
[48] Inducible nucleotide excision repair (NER) of UV-induced cyclobutane pyrimidine dimers in the cell cycle of the budding yeast Saccharomyces cerevisiae : evidence that inducible NER is confined to the G1 phase of the mitotic cell cycle
A. D. Scott
R. Waters
Molecular and General Genetics MGG, 1997, 254 : 43 - 53

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