IRREVERSIBLE PLATELET FIBRINOGEN INTERACTIONS OCCUR INDEPENDENTLY OF FIBRINOGEN ALPHA-CHAIN DEGRADATION AND ARE NOT MEDIATED BY INTACT PLATELET MEMBRANE GPIIB-IIIA COMPLEXES

被引：0

作者：

PEERSCHKE, EIB ^{[1
]}

机构：

[1] SUNY STONY BROOK,DEPT PATHOL,STONY BROOK,NY 11754

来源：

FEDERATION PROCEEDINGS | 1987年 / 46卷 / 03期

关键词：

D O I：

暂无

中图分类号：

Q [生物科学];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

引用

页码：721 / 721

页数：1

共 50 条

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[43] A PEPTIDE CORRESPONDING TO GPIIB-ALPHA 300-312, A PRESUMPTIVE FIBRINOGEN GAMMA-CHAIN BINDING-SITE ON THE PLATELET INTEGRIN GPIIB/IIIA, INHIBITS THE ADHESION OF PLATELETS TO AT LEAST 4 ADHESIVE LIGANDS
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[45] Direct binding of the platelet integrin alpha(IIb)beta(3) (GPIIb-IIIa) to talin - Evidence that interaction is mediated through the cytoplasmic domains of both alpha(IIb) and beta(3)
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[46] Direct binding of the platelet integrin alpha(IIb)beta(3) (GPIIb-IIIa) to talin: Evidence that interaction is mediated through the cytoplasmic domains of both alpha(IIb) and beta(3).
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[47] ENDOCYTOSIS OF FIBRINOGEN INTO MEGAKARYOCYTE AND PLATELET ALPHA-GRANULES IS MEDIATED BY ALPHA(IIB)BETA(3) (GLYCOPROTEIN IIB-IIIA) (VOL 82, PG 135, 1993)
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[49] Fibrinogen receptor (GPIIb-IIIa) antagonists derived from 5,6-bicyclic templates.: Amidinoindoles, amidinoindazoles, and amidinobenzofurans containing the N-α-sulfonamide carboxylic acid function as potent platelet aggregation inhibitors
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[50] MAXIMAL FIBRINOGEN-PLATELET INTERACTIONS REQUIRE STERICALLY-RESTRICTED BIVALENT MOLECULES CONTAINING D-DOMAINS AND INTACT A-ALPHA-CHAINS
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