THE INTRINSIC INSTABILITY OF C-MYC MESSENGER-RNA AND ITS DOWN-REGULATION DURING DIFFERENTIATION OF C2 MYOBLASTS REQUIRES TRANSLATION OF PROTEIN-CODING SEQUENCES

被引：0

作者：

RESTA, R ^{[1
]}

LEE, W ^{[1
]}

机构：

[1] UNIV PENN,DEPT MED,PHILADELPHIA,PA 19104

来源：

CLINICAL RESEARCH | 1991年 / 39卷 / 02期

关键词：

D O I：

暂无

中图分类号：

R-3 [医学研究方法]; R3 [基础医学];

学科分类号：

1001 ;

摘要：

引用

页码：A318 / A318

页数：1

共 43 条

[31] Inactivation of a c-Myb/estrogen receptor fusion protein in transformed primary cells leads to granulocyte/macrophage differentiation and down regulation of c-kit but not c-myc or cdc2
Annette Hogg
Sabine Schirm
Hideki Nakagoshi
Paul Bartley
Shunsuke Ishii
J Michael Bishop
Thomas J Gonda
Oncogene, 1997, 15 : 2885 - 2898
[32] Inactivation of a c-Myb/estrogen receptor fusion protein in transformed primary cells leads to granulocyte/macrophage differentiation and down regulation of c-kit but not c-myc or cdc2
Hogg, A
Schirm, S
Nakagoshi, H
Bartley, P
Ishii, S
Bishop, JM
Gonda, TJ
ONCOGENE, 1997, 15 (24) : 2885 - 2898
[33] EXPRESSION OF MESSENGER-RNA SPECIES CODING FOR A MR-43,000 PEPTIDE ASSOCIATED WITH FERRITIN IN HUMAN LEUKEMIA-K562 CELLS AND ITS DOWN REGULATION DURING DIFFERENTIATION
SHTERMAN, N
KUPFER, B
MOROZ, C
CANCER RESEARCH, 1989, 49 (18) : 5033 - 5036
[34] TUMOR-NECROSIS-FACTOR-ALPHA DOWN-REGULATES C-MYC MESSENGER-RNA EXPRESSION AND INDUCES INVITRO MONOCYTIC DIFFERENTIATION IN FRESH BLAST CELLS FROM PATIENTS WITH ACUTE MYELOBLASTIC-LEUKEMIA
BERGAMASCHI, G
CARLOSTELLA, C
CAZZOLA, M
DEFAZIO, P
PEDRAZZOLI, P
PEVERALI, FA
DELLAVALLE, G
LEUKEMIA, 1990, 4 (06) : 426 - 430
[35] TIS21BTG2/PC3 enhances down-regulation of c-Myc expression during all-trans-retinoic acid-induced differentiation of acute promyelocytic leukemia cells
Imran, Muhammad
Park, Tae Jun
Lim, In Kyoung
CANCER RESEARCH, 2010, 70
[36] PROTEIN-KINASE-C DOWN-REGULATION ENHANCES CAMP-MEDIATED INDUCTION OF UROKINASE-TYPE PLASMINOGEN-ACTIVATOR MESSENGER-RNA IN LLC-PK1 CELLS
ZIEGLER, A
KNESEL, J
FABBRO, D
NAGAMINE, Y
JOURNAL OF BIOLOGICAL CHEMISTRY, 1991, 266 (31) : 21067 - 21074
[37] DIFFERENTIAL REGULATION OF C-MYC AND TRANSFORMING GROWTH FACTOR-ALPHA MESSENGER-RNA EXPRESSION IN POORLY DIFFERENTIATED AND WELL-DIFFERENTIATED COLON-CARCINOMA CELLS DURING THE ESTABLISHMENT OF A QUIESCENT STATE
MULDER, KM
CANCER RESEARCH, 1991, 51 (09) : 2256 - 2262
[38] LINOLEIC-ACID AND ITS METABOLITES, HYDROPEROXYOCTADECADIENOIC ACIDS, STIMULATE C-FOS, C-JUN, AND C-MYC MESSENGER-RNA EXPRESSION, MITOGEN-ACTIVATED PROTEIN-KINASE ACTIVATION, AND GROWTH IN RAT AORTIC SMOOTH-MUSCLE CELLS
RAO, GN
ALEXANDER, RW
RUNGE, MS
JOURNAL OF CLINICAL INVESTIGATION, 1995, 96 (02): : 842 - 847
[39] CD2+-INDUCED C-MYC MESSENGER-RNA ACCUMULATION IN NRK-49F CELLS IS BLOCKED BY THE PROTEIN-KINASE INHIBITOR H7 BUT NOT BY HA1004, INDICATING THAT PROTEIN-KINASE-C IS A MEDIATOR OF THE RESPONSE
TANG, N
ENGER, MD
TOXICOLOGY, 1993, 81 (02) : 155 - 164
[40] Down regulation of NM23.H1, NM23.H2 and c-myc genes during differentiation induced by 1,25 dihydroxyvitamin D-3
Caligo, MA
Cipollini, G
Petrini, M
Valentini, P
Bevilacqua, G
LEUKEMIA RESEARCH, 1996, 20 (02) : 161 - 167

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