MEMBRANE COMPLEMENT RECEPTOR TYPE-3 (CR3) HAS LECTIN-LIKE PROPERTIES ANALOGOUS TO BOVINE CONGLUTININ AND FUNCTIONS AS A RECEPTOR FOR ZYMOSAN AND RABBIT ERYTHROCYTES AS WELL AS A RECEPTOR FOR IC3B

被引：0

作者：

ROSS, GD

CAIN, JA

LACHMANN, PJ

机构：

[1] MRC,MECHANISMS TUMOR IMMUNITY UNIT,CAMBRIDGE,ENGLAND

[2] UNIV N CAROLINA,DEPT MICROBIOL & IMMUNOL,CHAPEL HILL,NC 27514

来源：

JOURNAL OF IMMUNOLOGY | 1985年 / 134卷 / 05期

关键词：

D O I：

暂无

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：3307 / 3315

页数：9

共 48 条

[41] The role of iC3b opsonization, C5a receptor and CD14 in E-coli-induced upregulation of leukocyte CD11b (CR3), phagocytosis and oxidative burst in human whole blood
Christiansen, D
Brekke, OL
Fure, H
Fung, M
Mollnes, TE
MOLECULAR IMMUNOLOGY, 2004, 41 (2-3) : 221 - 222
[42] MODULATION OF COMPLEMENT RECEPTORS OF A HUMAN MONOCYTE CELL-LINE, U-937, DURING INCUBATION WITH PHORBOL-MYRISTATE ACETATE - EXPRESSION OF AN IC3B-SPECIFIC RECEPTOR (CR3)
GILBERT, D
PEULVE, P
DAVEAU, M
RIPOCHE, J
FONTAINE, M
EUROPEAN JOURNAL OF IMMUNOLOGY, 1985, 15 (10) : 986 - 991
[43] Kinetic analysis of the interactions of complement receptor 2 (CR2, CD21) with its ligands C3d, iC3b, and the EBV glycoprotein gp350/220
Sarrias, MR
Franchini, S
Canziani, G
Argyropoulos, E
Moore, WT
Sahu, A
Lambris, JD
JOURNAL OF IMMUNOLOGY, 2001, 167 (03): : 1490 - 1499
[44] MUTAGENESIS OF RESIDUES IN THE REGION OF HUMAN-COMPLEMENT COMPONENT C3 THAT CORRESPONDS TO THE PROPOSED BINDING-SITE FOR COMPLEMENT RECEPTOR 2 (CR-2, CD21) DOES NOT ABOLISH BINDING OF IC3B OR C3DG TO CR-2
DIEFENBACH, RJ
ISENMAN, DE
FASEB JOURNAL, 1994, 8 (05): : A766 - A766
[45] MUTATION OF RESIDUES IN THE C3DG REGION OF HUMAN-COMPLEMENT COMPONENT C3 CORRESPONDING TO A PROPOSED BINDING-SITE FOR COMPLEMENT RECEPTOR-TYPE-2 (CR-2, CD21) DOES NOT ABOLISH BINDING OF IC3B OR C3DG TO CR-2
DIEFENBACH, RJ
ISENMAN, DE
JOURNAL OF IMMUNOLOGY, 1995, 154 (05): : 2303 - 2320
[46] The β-glucan-binding lectin site of mouse CR3 (CD11b/CD18) and its function in generating a primed state of the receptor that mediates cytotoxic activation in response to iC3b-opsonized target cells
Xia, Y
Vetvicka, V
Yan, J
Hanikyrová, M
Mayadas, T
Ross, GD
JOURNAL OF IMMUNOLOGY, 1999, 162 (04): : 2281 - 2290
[47] Characterization of hyaluronan-binding proteins on guinea pig polymorphonuclear leukocytes: Possible involvement of complement receptor type 3 (CR3, CD11b/CD18) in the hyaluronan-leukocyte interaction
Nochi, H
Shinomiya, T
Tamoto, K
JOURNAL OF BIOCHEMISTRY, 2006, 139 (01): : 59 - 70
[48] Soluble beta-glucan polysaccharide binding to the lectin site of neutrophil or natural killer cell complement receptor type 3 (CD11b/CD18) generates a primed state of the receptor capable of mediating cytotoxicity of iC3b-opsonized target cells
Vetvicka, V
Thornton, BP
Ross, GD
JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (01): : 50 - 61

← 1 2 3 4 5 →