The transcription factor IRF1 dictates the IL-21-dependent anticancer functions of TH9 cells

被引:0
|
作者
Frédérique Végran
Hélène Berger
Romain Boidot
Grégoire Mignot
Mélanie Bruchard
Magalie Dosset
Fanny Chalmin
Cédric Rébé
Valentin Dérangère
Bernhard Ryffel
Masashi Kato
Armelle Prévost-Blondel
François Ghiringhelli
Lionel Apetoh
机构
[1] INSERM,Department of Occupational and Environmental Health
[2] U866,undefined
[3] Faculté de Médecine,undefined
[4] Université de Bourgogne,undefined
[5] Centre Georges François Leclerc,undefined
[6] CNRS and University,undefined
[7] UMR7355,undefined
[8] Institute of Infectious Disease and Molecular Medicine,undefined
[9] University of Cape Town,undefined
[10] Nagoya University Graduate School of Medicine,undefined
[11] CNRS,undefined
[12] UMR 8104,undefined
[13] INSERM,undefined
[14] U1016,undefined
[15] Paris Descartes University,undefined
[16] Cochin Institute,undefined
来源
Nature Immunology | 2014年 / 15卷
关键词
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中图分类号
学科分类号
摘要
TH9 cells are associated with autoinflammatory diseases. Apetoh and colleagues demonstrate a molecular pathway which converts TH9 cells into potent anticancer cells.
引用
收藏
页码:758 / 766
页数:8
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