The transcription factor IRF1 dictates the IL-21-dependent anticancer functions of TH9 cells

被引：0

作者：

Frédérique Végran

Hélène Berger

Romain Boidot

Grégoire Mignot

Mélanie Bruchard

Magalie Dosset

Fanny Chalmin

Cédric Rébé

Valentin Dérangère

Bernhard Ryffel

Masashi Kato

Armelle Prévost-Blondel

François Ghiringhelli

Lionel Apetoh

机构：

[1] INSERM,Department of Occupational and Environmental Health

[2] U866,undefined

[3] Faculté de Médecine,undefined

[4] Université de Bourgogne,undefined

[5] Centre Georges François Leclerc,undefined

[6] CNRS and University,undefined

[7] UMR7355,undefined

[8] Institute of Infectious Disease and Molecular Medicine,undefined

[9] University of Cape Town,undefined

[10] Nagoya University Graduate School of Medicine,undefined

[11] CNRS,undefined

[12] UMR 8104,undefined

[13] INSERM,undefined

[14] U1016,undefined

[15] Paris Descartes University,undefined

[16] Cochin Institute,undefined

来源：

Nature Immunology | 2014年 / 15卷

关键词：

D O I：

暂无

中图分类号：

学科分类号：

摘要：

TH9 cells are associated with autoinflammatory diseases. Apetoh and colleagues demonstrate a molecular pathway which converts TH9 cells into potent anticancer cells.

引用

页码：758 / 766

页数：8

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