The histone H4 lysine 16 acetyltransferase hMOF regulates the outcome of autophagy

被引：0
|
作者：
Jens Füllgrabe
Melinda A. Lynch-Day
Nina Heldring
Wenbo Li
Robert B. Struijk
Qi Ma
Ola Hermanson
Michael G. Rosenfeld
Daniel J. Klionsky
Bertrand Joseph
机构：
[1] Cancer Centrum Karolinska,Department of Oncology Pathology
[2] Karolinska Institutet,Life Sciences Institute and Departments of Molecular
[3] Stockholm 17176,Department of Neuroscience
[4] Sweden,Department of Medicine
[5] Cellular and Developmental Biology and Biological Chemistry,undefined
[6] University of Michigan,undefined
[7] Ann Arbor,undefined
[8] Karolinska Institutet,undefined
[9] Stockholm 17177,undefined
[10] Sweden,undefined
[11] Howard Hughes Medical Institute,undefined
[12] School of Medicine,undefined
[13] University of California,undefined
[14] San Diego,undefined
[15] La Jolla,undefined
[16] California 92093,undefined
[17] USA,undefined
[18] Graduate Program in Bioinformatics and Systems Biology,undefined
[19] University of California,undefined
[20] San Diego,undefined
[21] La Jolla,undefined
[22] California 92093,undefined
[23] USA,undefined
来源：
Nature | 2013年 / 500卷
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摘要：
Induction of autophagy is coupled to reduction of histone H4 lysine 16 acetylation through downregulation of the histone acetyltransferase hMOF, showing that histone modifications regulate the outcome of autophagy.
引用
收藏
页码：468 / 471
页数：3
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