Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells

被引：0

作者：

Jhimmy Talbot

Raphael S. Peres

Larissa G. Pinto

Rene D. R. Oliveira

Kalil A. Lima

Paula B. Donate

Jaqueline R. Silva

Bernard Ryffel

Thiago M. Cunha

José C. Alves-Filho

Foo Y. Liew

Paulo Louzada-Junior

Fernando de Queiroz Cunha

机构：

[1] Ribeirao Preto Medical School,Department of Pharmacology

[2] University of Sao Paulo,Department of Immunology

[3] Ribeirao Preto Medical School,Division of Clinical Immunology

[4] University of São Paulo,Institute of Infectious Disease and Molecular Medicine

[5] Ribeirao Preto Medical School,Division of Immunology, Infection and Inflammation, Glasgow Biomedical Research Centre

[6] University of São Paulo,undefined

[7] Université Orleans and Centre National de la Recherche Scientifique,undefined

[8] Molecular Immunology,undefined

[9] UMR7355,undefined

[10] INEM,undefined

[11] UCT,undefined

[12] University of Glasgow,undefined

[13] School of Biology and Basic Medical Sciences,undefined

[14] Soochow University,undefined

来源：

Arthritis Research & Therapy | / 20卷

关键词：

Rheumatoid arthritis; Cigarette smoke; Inflammation; Polycyclic aromatic hydrocarbons; Th17;

D O I：

暂无

中图分类号：

学科分类号：

摘要：

引用

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