Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells

被引：0
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作者：
Jhimmy Talbot
Raphael S. Peres
Larissa G. Pinto
Rene D. R. Oliveira
Kalil A. Lima
Paula B. Donate
Jaqueline R. Silva
Bernard Ryffel
Thiago M. Cunha
José C. Alves-Filho
Foo Y. Liew
Paulo Louzada-Junior
Fernando de Queiroz Cunha
机构：
[1] Ribeirao Preto Medical School,Department of Pharmacology
[2] University of Sao Paulo,Department of Immunology
[3] Ribeirao Preto Medical School,Division of Clinical Immunology
[4] University of São Paulo,Institute of Infectious Disease and Molecular Medicine
[5] Ribeirao Preto Medical School,Division of Immunology, Infection and Inflammation, Glasgow Biomedical Research Centre
[6] University of São Paulo,undefined
[7] Université Orleans and Centre National de la Recherche Scientifique,undefined
[8] Molecular Immunology,undefined
[9] UMR7355,undefined
[10] INEM,undefined
[11] UCT,undefined
[12] University of Glasgow,undefined
[13] School of Biology and Basic Medical Sciences,undefined
[14] Soochow University,undefined
来源：
Arthritis Research & Therapy | / 20卷
关键词：
Rheumatoid arthritis; Cigarette smoke; Inflammation; Polycyclic aromatic hydrocarbons; Th17;
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