Dual effects of carbon monoxide on pericytes and neurogenesis in traumatic brain injury

被引：0

作者：

Yoon Kyung Choi

Takakuni Maki

Emiri T Mandeville

Seong-Ho Koh

Kazuhide Hayakawa

Ken Arai

Young-Myeong Kim

Michael J Whalen

Changhong Xing

Xiaoying Wang

Kyu-Won Kim

Eng H Lo

机构：

[1] Massachusetts General Hospital,Department of Radiology

[2] Harvard Medical School,Department of Neurology

[3] Massachusetts General Hospital,Department of Neurology

[4] Harvard Medical School,Department of Molecular and Cellular Biochemistry

[5] Hanyang University College of Medicine,Department of Pediatrics

[6] School of Medicine,undefined

[7] Kangwon National University,undefined

[8] Chuncheon,undefined

[9] Massachusetts General Hospital,undefined

[10] Harvard Medical School,undefined

[11] SNU–Harvard NeuroVascular Protection Research Center,undefined

[12] Research Institute of Pharmaceutical Sciences,undefined

[13] College of Pharmacy,undefined

[14] Seoul National University,undefined

来源：

Nature Medicine | 2016年 / 22卷

关键词：

D O I：

暂无

中图分类号：

学科分类号：

摘要：

In a mouse model of traumatic brain injury, treatment with a carbon-monoxide-releasing molecule is shown to reduce pericyte cell death and promote neurogenesis, leading to an amelioration of neurological deficits.

引用

页码：1335 / 1341

页数：6

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