Dual effects of carbon monoxide on pericytes and neurogenesis in traumatic brain injury

被引:0
|
作者
Yoon Kyung Choi
Takakuni Maki
Emiri T Mandeville
Seong-Ho Koh
Kazuhide Hayakawa
Ken Arai
Young-Myeong Kim
Michael J Whalen
Changhong Xing
Xiaoying Wang
Kyu-Won Kim
Eng H Lo
机构
[1] Massachusetts General Hospital,Department of Radiology
[2] Harvard Medical School,Department of Neurology
[3] Massachusetts General Hospital,Department of Neurology
[4] Harvard Medical School,Department of Molecular and Cellular Biochemistry
[5] Hanyang University College of Medicine,Department of Pediatrics
[6] School of Medicine,undefined
[7] Kangwon National University,undefined
[8] Chuncheon,undefined
[9] Massachusetts General Hospital,undefined
[10] Harvard Medical School,undefined
[11] SNU–Harvard NeuroVascular Protection Research Center,undefined
[12] Research Institute of Pharmaceutical Sciences,undefined
[13] College of Pharmacy,undefined
[14] Seoul National University,undefined
来源
Nature Medicine | 2016年 / 22卷
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学科分类号
摘要
In a mouse model of traumatic brain injury, treatment with a carbon-monoxide-releasing molecule is shown to reduce pericyte cell death and promote neurogenesis, leading to an amelioration of neurological deficits.
引用
收藏
页码:1335 / 1341
页数:6
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