CXCR7 suppression modulates macrophage phenotype and function to ameliorate post-myocardial infarction injury

被引：1
|
作者：
Junshi Zhang
Ying Zhang
Shifeng Xin
Min Wu
Yaling Zhang
Lihua Sun
机构：
[1] The First Affiliated Hospital of Xinjiang Medical University,Department of Hypertension, Heart Center
[2] The Fifth Affiliated Hospital of Xinjiang Medical University,Department of Cardiology
来源：
Inflammation Research | 2020年 / 69卷
关键词：
CXCR7; Macrophage; Chemotaxis; Myocardial infarction (MI); Extracellular signal-regulated kinase (ERK);
D O I：
暂无
中图分类号：
学科分类号：
摘要：
引用
收藏
页码：523 / 532
页数：9
相关论文
共 50 条
  • [31] Electrospun extracellular matrix scaffold improves cardiac structure and function post-myocardial infarction
    Hayam, Rotem
    Ertracht, Offir
    Zahran, Sharbel
    Baruch, Limor
    Atar, Shaul
    Machluf, Marcelle
    POLYMERS FOR ADVANCED TECHNOLOGIES, 2022, 33 (11) : 3822 - 3831
  • [32] Left ventricular structure and function post-myocardial infarction in mice heterozygous for MnSOD deficiency
    Trueblood, NA
    Ngoy, S
    Pligavko, C
    Inscore, P
    Huang, TT
    Epstein, CJ
    Apstein, CS
    Colucci, WS
    Sawyer, DB
    FASEB JOURNAL, 2001, 15 (04): : A479 - A479
  • [33] Granulocyte-macrophage colony-stimulating factor inducer accelerates post-myocardial infarction remodeling
    Yuichiro, M
    Toshihisa, A
    Tsutomu, Y
    Satoshi, O
    JOURNAL OF CARDIAC FAILURE, 2004, 10 (05) : S182 - S182
  • [34] Thymic stromal lymphopoietin modulates T cell response and improves cardiac repair post-myocardial infarction
    Wang, Xuhong
    Zheng, Qi
    Zha, Lingfeng
    Zhang, Lingxue
    Huang, Mingkai
    Zhang, Si
    Zhang, Xuzhe
    Li, Qinlin
    Chen, Xinglin
    Xia, Ni
    Zhang, Min
    Lv, Bingjie
    Jiao, Jiao
    Lu, Yuzhi
    Gu, Muyang
    Yang, Fen
    Li, Jingyong
    Li, Nana
    Cheng, Xiang
    Zhou, Zihua
    Tang, Tingting
    FRONTIERS IN IMMUNOLOGY, 2024, 15
  • [35] The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction
    Li, Jun
    Li, Ruoshui
    Tuleta, Izabela
    Hernandez, Silvia C.
    Humeres, Claudio
    Hanna, Anis
    Chen, Bijun
    Frangogiannis, Nikolaos G.
    FASEB JOURNAL, 2022, 36 (07):
  • [36] Matrix metalloproteinase-9 deletion shifts macrophage polarization towards M2 phenotype in aged left ventricles post-myocardial infarction
    Yabluchanskiy, A.
    Ma, Y.
    Deleon-Pennell, K.
    Jin, Y-F.
    Lindsey, M.
    CARDIOVASCULAR RESEARCH, 2014, 103
  • [37] Stimulating Myocardial Regeneration with Periostin Peptide in Large Mammals Improves Function Post-Myocardial Infarction but Increases Myocardial Fibrosis
    Ladage, Dennis
    Yaniz-Galende, Elisa
    Rapti, Kleopatra
    Ishikawa, Kiyotake
    Tilemann, Lisa
    Shapiro, Scott
    Takewa, Yoshiaki
    Muller-Ehmsen, Jochen
    Schwarz, Martin
    Garcia, Mario J.
    Sanz, Javier
    Hajjar, Roger J.
    Kawase, Yoshiaki
    PLOS ONE, 2013, 8 (06):
  • [38] Hypertrophic preconditioning attenuates post-myocardial infarction injury through deacetylation of isocitrate dehydrogenase 2
    Lei-lei Ma
    Fei-juan Kong
    Yuan-ji Ma
    Jun-jie Guo
    Shi-jun Wang
    Zheng Dong
    Ai-jun Sun
    Yun-zeng Zou
    Jun-bo Ge
    Acta Pharmacologica Sinica, 2021, 42 : 2004 - 2015
  • [39] Gender and hypertension independently influence susceptability to ischemia-reperfusion injury post-myocardial infarction
    Jain, M
    Ngoy, S
    Eberli, FR
    CIRCULATION, 1998, 98 (17) : 638 - 638
  • [40] Differential Response to Injury in Fetal and Adolescent Sheep Hearts in the Immediate Post-myocardial Infarction Period
    Lock, Mitchell C.
    Darby, Jack R. T.
    Soo, Jia Yin
    Brooks, Doug A.
    Perumal, Sunthara Rajan
    Selvanayagam, Joseph B.
    Seed, Mike
    Macgowan, Christopher K.
    Porrellov, Enzo R.
    Tellam, Ross L.
    Morrison, Janna L.
    FRONTIERS IN PHYSIOLOGY, 2019, 10
12345