HAM/TSP-derived HTLV-1-infected T cell lines promote morphological and functional changes in human astrocytes cell lines: possible role in the enhanced T cells recruitment into Central Nervous System

被引：0

作者：

Eduardo Samo Gudo

Suse Dayse Silva-Barbosa

Leandra Linhares-Lacerda

Marcelo Ribeiro-Alves

Suzana Corte Real

Dumith Chequer Bou-Habib

Wilson Savino

机构：

[1] National Institute of Health,

[2] Ministry of Health,undefined

[3] Laboratory on Thymus Research,undefined

[4] Oswaldo Cruz Institute,undefined

[5] Oswaldo Cruz Foundation,undefined

[6] National Cancer Institute,undefined

[7] Laboratory of Structural Biology,undefined

[8] Oswaldo Cruz Institute,undefined

[9] Oswaldo Cruz Foundation,undefined

来源：

Virology Journal | / 12卷

关键词：

Astrocytes; HTLV-1-infected lymphocytes; Extracellular matrix; Chemokines; Integrins;

D O I：

暂无

中图分类号：

学科分类号：

摘要：

引用

共 19 条

[11] FREQUENT CLONAL PROLIFERATION OF HUMAN T-CELL LEUKEMIA-VIRUS TYPE-1 (HTLV-1)-INFECTED T-CELLS IN HTLV-1-ASSOCIATED MYELOPATHY (HAM-TSP)
FURUKAWA, Y
FUJISAWA, J
OSAME, M
TOITA, M
SONODA, S
KUBOTA, R
IJICHI, S
YOSHIDA, M
BLOOD, 1992, 80 (04) : 1012 - 1016
[12] The involvement of integrins/ligands signaling in HTLVI production from HTLV-I-infected T cell lines derived from patients with HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP)
Fukushima, Naomi
Nakamura, Tatsufumi
Nishiura, Yoshihiro
Ida, Hiroaki
Aramaki, Toshiyuki
Eguchi, Katsumi
AIDS RESEARCH AND HUMAN RETROVIRUSES, 2007, 23 (04) : 617 - 618
[13] The down-regulation of HTLV-I production by anti-integrin blocking antibodies in HTLV-I-infected T cell lines derived from patients with HTLV-I associated myelopathy tropical spastic paraparesis (HAM/TSP)
Fukushima, N.
Nakamura, T.
Nishiura, Y.
Ida, H.
Yamada, Y.
Kohno, S.
Eguchi, K.
JOURNAL OF NEUROIMMUNOLOGY, 2006, 178 : 257 - 257
[14] Potential role of HTLV-1 Tax-specific cytotoxic t lymphocytes expressing a unique t-cell receptor to promote inflammation of the central nervous system in myelopathy associated with HTLV-1
Tanaka, Yukie
Sato, Tomoo
Yagishita, Naoko
Yamauchi, Junji
Araya, Natsumi
Aratani, Satoko
Takahashi, Katsunori
Kunitomo, Yasuo
Nagasaka, Misako
Kanda, Yoshinobu
Uchimaru, Kaoru
Morio, Tomohiro
Yamano, Yoshihisa
FRONTIERS IN IMMUNOLOGY, 2022, 13
[15] Activation of the PI3K-Akt pathway by human T cell leukemia virus type 1 (HTLV-1) oncoprotein Tax increases Bcl3 expression, which is associated with enhanced growth of HTLV-1-infected T cells
Saito, Kousuke
Saito, Mineki
Taniura, Naoko
Okuwa, Takako
Ohara, Yoshiro
VIROLOGY, 2010, 403 (02) : 173 - 180
[16] Rapid syncytium formation between human T-cell leukaemia virus type-I (HTLV-I)-infected T-cells and human nervous system cells: A possible implication for tropical spastic paraparesis HTLV-I associated myelopathy
Mor-Vaknin, N
Turgeman, H
Torgeman, A
Wolfson, M
Huleihel, M
Aboud, M
CELL BIOLOGY INTERNATIONAL, 1998, 22 (02) : 95 - 103
[17] Host T cells are the main producers of IL-17 within the central nervous system during initiation of experimental autoimmune encephalomyelitis of Th1 cell lines
Lees, Jason R.
Iwakura, Yoichiro
Russell, John H.
JOURNAL OF IMMUNOLOGY, 2008, 180 (12): : 8066 - 8072
[18] Interactions between brain endothelial cells and human T-cell leukemia virus type 1-infected lymphocytes: Mechanisms of viral entry into the central nervous system
Romero, IA
Prevost, MC
Perret, E
Adamson, P
Greenwood, J
Couraud, PO
Ozden, S
JOURNAL OF VIROLOGY, 2000, 74 (13) : 6021 - 6030
[19] FUSION OF HUMAN B-CELL LINES WITH HIV-1 ENVELOPE-EXPRESSING T-CELLS IS ENHANCED BY ANTIGEN-SPECIFIC IG RECEPTORS - POSSIBLE MECHANISM FOR ELIMINATION OF GP120-SPECIFIC B-CELLS INVIVO
GOLDING, H
DIMITROV, DS
BLACKBURN, R
MANISCHEWITZ, J
BLUMENTHAL, R
GOLDING, B
JOURNAL OF IMMUNOLOGY, 1993, 150 (06): : 2506 - 2516

← 1 2 →