Host immune defence, amyloid-β peptide and Alzheimer disease

被引：0
|
作者：
Todd E. Golde
机构：
[1] Center for Translational Research in Neurodegenerative Disease,Department of Neuroscience
[2] McKnight Brian Institute,undefined
[3] College of Medicine,undefined
[4] University of Florida,undefined
来源：
Nature Reviews Neurology | 2016年 / 12卷
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D O I：
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学科分类号：
摘要：
A recent study demonstrates that amyloid-β (Aβ) can function as an antimicrobial peptide, and additional data show that bacteria and yeast can seed Aβ deposition into amyloid. These data suggest a complex interplay between the normal function of Aβ, its accumulation in the brain, and host immune defence.
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页码：433 / 434
页数：1
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