microRNA regulation of mammalian target of rapamycin expression and activity controls estrogen receptor function and RAD001 sensitivity

被引：0

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作者：

Elizabeth C Martin

Lyndsay V Rhodes

Steven Elliott

Adrienne E Krebs

Kenneth P Nephew

Erik K Flemington

Bridgette M Collins-Burow

Matthew E Burow

机构：

[1] Tulane University,Department of Medicine

[2] Tulane University,Section of Hematology and Medical Oncology

[3] Tulane University,Department of Pharmacology

[4] Tulane University,The Center for Bioenvironmental Research

[5] Indiana University School of Medicine,Department of Pathology

来源：

Molecular Cancer | / 13卷

关键词：

miR-155; mTOR; breast cancer; miRNA; Estrogen receptor;

D O I：

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[41] Insulin-like growth factor-I inhibits progesterone receptor expression in breast cancer cells via the phosphatidylinositol 3-kinase/akt/mammalian target of rapamycin pathway: Progesterone receptor as a potential indicator of growth factor activity in breast cancer
Cui, XJ
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MOLECULAR ENDOCRINOLOGY, 2003, 17 (04) : 575 - 588
[42] Everolimus (RAD001) combined with programmed death-1 (PD-1) blockade enhances radiosensitivity of cervical cancer and programmed death-ligand 1 (PD-L1) expression by blocking the phosphoinositide 3-kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR)/S6 kinase 1 (S6K1) pathway
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[43] Insulin-like growth factor-1 inhibits progesterone receptor expression in breast cancer cells via the phosphatidylinositol 3-kinase/akt/mammalian target of rapamycin pathway: Progesterone receptor as a potential indication of growth factor activity in breast cancer (vol 17, pg 575, 2003)
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