Inhibition of mTORC1 signaling protects kidney from irradiation-induced toxicity via accelerating recovery of renal stem-like cells

被引：0

作者：

Lijian Shao

Wuping Yang

Rui Xu

Shuqin Zhu

Yanqiu Huang

Huan Li

Xincheng Wu

Mengzhen Yue

Xiaoliang Xiong

Xiaowen Chen

Bohai Kuang

Guangqin Fan

Qingxian Zhu

Huihong Zeng

机构：

[1] Nanchang University,Jiangxi Provincial Key Laboratory of Preventive Medicine

[2] Nanchang University,School of Public Health

[3] Basic Medical School of Nanchang University,Department of Histology and Embryology

[4] The Fifth Affiliated Hospital of Sun Yat-sen University,Department of Pathology

[5] The First Affiliated Hospital of Nanchang University,Department of Pathology

[6] Basic Medical School of Nanchang University,Department of Medical Genetics and Cell Biology

[7] Basic Medical School of Nanchang University,undefined

来源：

Stem Cell Research & Therapy | / 9卷

关键词：

Irradiation; Renal damage; mTOR complex 1; Rapamycin; Apoptosis;

D O I：

暂无

中图分类号：

学科分类号：

摘要：

引用

共 18 条

[11] Food-derived antidepressant-like compound ergothioneine promotes neuronal differentiation via activating mTORC1 and neurotrophic factor signaling in neural stem cells.
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[12] Zerumbone protects HEK 293 cells from irradiation-induced DNA damage via activating Keap1/Nrf2/ARE pathway
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[13] Rg1 Protects Hematopoietic Stem Cells from LiCl-Induced Oxidative Stress via Wnt Signaling Pathway
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[14] Ebselen, Iron Uptake Inhibitor, Alleviates Iron Overload-Induced Senescence-Like Neuronal Cells SH-SY5Y via Suppressing the mTORC1 Signaling Pathway
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[15] Curcumin Suppresses Phthalate-Induced Metastasis and the Proportion of Cancer Stem Cell (CSC)-like Cells via the Inhibition of AhR/ERK/SK1 Signaling in Hepatocellular Carcinoma
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Lee, Jau-Nan
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[17] Silencing CCNG1 protects MPC-5 cells from high glucose-induced proliferation-inhibition and apoptosis-promotion via MDM2/p53 signaling pathway
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Yan, Rui
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Liu, Jun
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INTERNATIONAL UROLOGY AND NEPHROLOGY, 2020, 52 (03) : 581 - 593
[18] Silencing CCNG1 protects MPC-5 cells from high glucose-induced proliferation-inhibition and apoptosis-promotion via MDM2/p53 signaling pathway
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