Retention of GluR1 receptors by ARMS/Kidins220 protein results in changes in synaptic activity

被引：0

作者：

Arevalo, J. C.

Takahashi, T.

Wu, S. H.

Chao, M. V.

机构：

[1] NYU, Sch Med, Skirball Inst Biomol Med, Dept Cell Biol,Mol Neurobiol Program, New York, NY USA

[2] NYU, Sch Med, Dept Physiol & Neurosci, New York, NY USA

[3] Cold Spring Harbor Lab, Cold Spring Harbor, NY 11724 USA

来源：

JOURNAL OF NEUROCHEMISTRY | 2007年 / 101卷

关键词：

D O I：

暂无

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：48 / 49

页数：2

共 36 条

[21] Synaptic incorporation of AMPA receptors during LTP is controlled by a PKC phosphorylation site on GluR1
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[22] Ankyrin Repeat-Rich Membrane Spanning (ARMS)/Kidins220 Scaffold Protein Regulates Neuroblastoma Cell Proliferation through p21
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[23] NMDA induces long-term synaptic depression and dephosphorylation of the GluR1 subunit of AMPA receptors in hippocampus
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[24] Two mutations preventing PDZ-protein interactions of GluR1 have opposite effects on synaptic plasticity
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LEARNING & MEMORY, 2006, 13 (05) : 562 - 565
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MOLECULAR AND CELLULAR NEUROSCIENCE, 2002, 19 (02) : 138 - 151
[27] RARE GENETIC VARIANTS AND NOVEL MISSENSE CODING MUTATIONS IN THE NEUROTROPHIN SIGNALING PATHWAY GENES WITH FOCUS ON THE SCAFFOLD PROTEIN ARMS/KIDINS220 AND RHO-GEF TRIO ARE IMPLICATED IN PSYCHOSIS RISK
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[28] Protein kinases regulate the phosphorylation of the GluR1 subunit of AMPA receptors of spinal cord in rats following noxious stimulation
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MOLECULAR BRAIN RESEARCH, 2003, 118 (1-2): : 160 - 165
[29] PROTEIN KINASE C-DEPENDENT AND INDEPENDENT SIGNALING PATHWAYS REGULATE SYNAPTIC GluR1 AND GluR4 AMPAR SUBUNITS DURING IN VITRO CLASSICAL CONDITIONING
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NEUROSCIENCE, 2008, 156 (04) : 872 - 884
[30] A novel two-site enzyme immunoassay reveals the regional distributions of and developmental changes in GluR1 and NMDAR1 protein contents in the rat brain
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