Bcr-Abl-induced evasion from apoptosis: A mechanism for tumorigenicity

被引：0
|
作者：
Jagani, Z
Ghaffari, S
Song, K
Kitidis, C
Lodish, H
Khosravi-Far, R
机构：
[1] Harvard Univ, Sch Med, BIDMC, Boston, MA USA
[2] Mt Sinai Sch Med, Carl C Icahn Inst Gene Therapy & Mol Med, New York, NY USA
[3] Whitehead Inst Biomed Res, Cambridge, MA 02142 USA
来源：
MOLECULAR BIOLOGY OF THE CELL | 2004年 / 15卷
关键词：
D O I：
暂无
中图分类号：
Q2 [细胞生物学];
学科分类号：
071009 ; 090102 ;
摘要：
2071
引用
收藏
页码：375A / 375A
页数：1
相关论文
共 50 条
  • [1] FoxO tumor suppressors and BCR-ABL-induced leukemia: A matter of evasion of apoptosis
    Jagani, Zainab
    Singh, Amrik
    Khosravi-Far, Roya
    BIOCHIMICA ET BIOPHYSICA ACTA-REVIEWS ON CANCER, 2008, 1785 (01): : 63 - 84
  • [2] Molecular mechanisms of Bcr-Abl-induced oncogenesis
    Gishizky, ML
    CYTOKINES AND MOLECULAR THERAPY, 1996, 2 (04): : 251 - 261
  • [3] Proteasome Inhibition Causes Regression of Leukemia and Abrogates BCR-ABL-Induced Evasion of Apoptosis in Part through Regulation of Forkhead Tumor Suppressors
    Jagani, Zainab
    Song, Keli
    Kutok, Jeffery L.
    Dewar, M. Rajan
    Melet, Armelle
    Santos, Tanya
    Grassian, Alexandra
    Ghaffari, Saghi
    Wu, Catherine
    Ren, Ruibao
    Rodin, Heather Yeckes
    Miller, Kenneth
    Khosravi-Far, Roya
    CANCER RESEARCH, 2009, 69 (16) : 6546 - 6555
  • [4] Lipocalin 2 is required for BCR-ABL-induced tumorigenesis
    Leng, X.
    Lin, H.
    Ding, T.
    Wang, Y.
    Wu, Y.
    Klumpp, S.
    Sun, T.
    Zhou, Y.
    Monaco, P.
    Belmont, J.
    Aderem, A.
    Akira, S.
    Strong, R.
    Arlinghaus, R.
    ONCOGENE, 2008, 27 (47) : 6110 - 6119
  • [5] Lipocalin 2 is required for BCR-ABL-induced tumorigenesis
    X Leng
    H Lin
    T Ding
    Y Wang
    Y Wu
    S Klumpp
    T Sun
    Y Zhou
    P Monaco
    J Belmont
    A Aderem
    S Akira
    R Strong
    R Arlinghaus
    Oncogene, 2008, 27 : 6110 - 6119
  • [6] Evaluation of Extrinsic and Intrinsic Cues Involved in BCR-ABL-Induced Leukemogenesis
    Sontakke, Pallavi Suhas
    Groen, Richard W. J.
    Koczula, Katarzyna
    Jaques, Jennifer
    Yuan, Huipin
    Guenther, Ulrich
    Martens, Anton C.
    Vellenga, Edo
    Schuringa, Jan J.
    BLOOD, 2014, 124 (21)
  • [7] BCR-ABL-induced adhesion defects are tyrosine kinase-independent
    Wertheim, JA
    Forsythe, K
    Druker, BJ
    Hammer, D
    Boettiger, D
    Pear, WS
    BLOOD, 2002, 99 (11) : 4122 - 4130
  • [8] MAPK15 MEDIATES BCR-ABL-INDUCED AUTOPHAGY AND CELLULAR TRANSFORMATION
    Colecchia, David
    Chiariello, Mario
    ANTICANCER RESEARCH, 2014, 34 (10) : 5864 - 5864
  • [9] Absence of SKP2 expression attenuates BCR-ABL-induced myeloproliferative disease
    Agarwal, Anupriya
    Bumm, Thomas G. P.
    Corbin, Arnie S.
    O'Hare, Thomas
    Loriaux, Marc
    VanDyke, Jonathan
    Willis, Stephanie G.
    Deininger, Jutta
    Nakayama, Keiichi I.
    Druker, Brian J.
    Deininger, Michael W.
    BLOOD, 2008, 112 (05) : 1960 - 1970
  • [10] PTEN is a tumor suppressor in CML stem cells and BCR-ABL-induced leukemias in mice
    Peng, Cong
    Chen, Yaoyu
    Yang, Zhongfa
    Zhang, Haojian
    Osterby, Lori
    Rosmarin, Alan G.
    Li, Shaoguang
    BLOOD, 2010, 115 (03) : 626 - 635
12345