Loss of PRDX1 increases RAD51 Cys319 oxidation and decreases homologous recombination

被引：0

作者：

Skoko, John ^{[1
]}

Asan, Alp ^{[1
]}

Woodcock, Chen ^{[1
]}

Cao, Juxiang ^{[2
]}

Gaboriau, David ^{[3
]}

Paulsen, Candice ^{[4
]}

Attar, Myriam ^{[1
]}

Wingert, Bentley ^{[1
]}

Woodcock, Steven ^{[1
]}

Schulte, Jennifer ^{[2
]}

Ma, Hongqiang ^{[1
]}

Camacho, Carlos ^{[1
]}

Liu, Yang ^{[1
]}

Morrison, Ciaran ^{[5
]}

Carroll, Kate ^{[4
]}

Freeman, Bruce ^{[1
]}

Neumann, Carola ^{[1
]}

机构：

[1] Univ Pittsburgh, Pittsburgh, PA 15260 USA

[2] Med Univ South Carolina, Charleston, SC 29425 USA

[3] Imperial Coll London, London, England

[4] Scripps Res Inst, La Jolla, CA 92037 USA

[5] Natl Univ Ireland Galway, Galway, Ireland

来源：

FREE RADICAL BIOLOGY AND MEDICINE | 2018年 / 128卷

关键词：

D O I：

10.1016/j.freeradbiomed.2018.10.159

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

155

引用

页码：S73 / S74

页数：2

共 45 条

[31] Inhibition of homologous recombination by treatment with BVDU (brivudin) or by RAD51 silencing increases chromosomal damage induced by bleomycin in mismatch repair-deficient tumour cells
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[32] MUTANT IDH1-DRIVEN CELLULAR TRANSFORMATION INCREASES RAD51-MEDIATED HOMOLOGOUS RECOMBINATION AND TEMOZOLOMIDE RESISTANCE
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[33] Mutant IDH1-Driven Cellular Transformation Increases RAD51-Mediated Homologous Recombination and Temozolomide Resistance
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[36] Role of the insulin-like growth factor I insulin receptor substrate 1 axis in Rad51 trafficking and DNA repair by homologous recombination
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[37] Rad18 Regulates BRCA2, FANCD2, and Rad51 to Repair Topoisomerase 1 Inhibitor-Induced DNA Lesions through Homologous Recombination
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[38] MUTANT IDH1-DRIVEN CELLULAR TRANSFORMATION INCREASES RAD51-MEDIATED HOMOLOGOUS RECOMBINATION AND TEMOZOLOMIDE (TMZ) RESISTANCE.
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[39] Repression of LSD1 potentiates homologous recombination-proficient ovarian cancer to PARP inhibitors through down-regulation of BRCA1/2 and RAD51
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[40] Repression of LSD1 sensitizes homologous recombination-proficient ovarian cancer to PARP inhibitors through down-regulation of BRCA1/2 and RAD51
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