Reduced tyrosine phosphorylation of P210BCR-ABL and lack of STAT5 activation in BCR-ABL-transduced IL-3-/- primitive hematopoietic cells.

被引:0
|
作者
Jiang, XY [1 ]
Yip, C [1 ]
Eisterer, W [1 ]
Eaves, A [1 ]
Eaves, C [1 ]
机构
[1] British Columbia Canc Agcy, Terry Fox Lab, Vancouver, BC V5Z 1L3, Canada
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
395
引用
下载
收藏
页码:92A / 92A
页数:1
相关论文
共 50 条
  • [21] p210BCR-ABL domain-specific control of lineage reprogramming and growth factor-independence exhibited by transduced human CD34+ cord blood cells.
    Chalandon, Y
    Jiang, XY
    Loutet, S
    Eaves, A
    Eaves, C
    BLOOD, 2001, 98 (11) : 761A - 761A
  • [22] Inhibition of Bcr–Abl kinase activity by PD180970 blocks constitutive activation of Stat5 and growth of CML cells
    Mei Huang
    Jay F Dorsey
    PK Epling-Burnette
    Ramadevi Nimmanapalli
    Terry H Landowski
    Linda B Mora
    Guilian Niu
    Dominic Sinibaldi
    Fanqi Bai
    Alan Kraker
    Hua Yu
    Lynn Moscinski
    Sheng Wei
    Julie Djeu
    William S Dalton
    Kapil Bhalla
    Thomas P Loughran
    Jie Wu
    Richard Jove
    Oncogene, 2002, 21 : 8804 - 8816
  • [23] Tyrosine phosphorylation of p120cbl in BCR/abl transformed hematopoietic cells mediates enhanced association with phosphatidylinositol 3-kinase
    Suresh K Jain
    Wallace Y Langdon
    Lyuba Varticovski
    Oncogene, 1997, 14 : 2217 - 2228
  • [24] Tyrosine phosphorylation of p120(cbl) in BCR/abl transformed hematopoietic cells mediates enhanced association with phosphatidylinositol 3-kinase
    Jain, SK
    Langdon, WY
    Varticovski, L
    ONCOGENE, 1997, 14 (18) : 2217 - 2228
  • [25] A 41-KILODALTON PROTEIN IS A POTENTIAL SUBSTRATE FOR THE P210BCR-ABL PROTEIN-TYROSINE KINASE IN CHRONIC MYELOGENOUS LEUKEMIA-CELLS
    FREED, E
    HUNTER, T
    MOLECULAR AND CELLULAR BIOLOGY, 1992, 12 (03) : 1312 - 1323
  • [26] IPO-trimethylation of histone H3-lysine9 associated with P210BCR-ABL tyrosine kinase of chronic myeloid leukaemia
    Mancini, Manuela
    Zuffa, Elisa
    Veljkovic, Nevena
    Brusa, Gianluca
    Corrado, Patrizia
    Corradi, Valentina
    Martinelli, Giovanni
    Barbieri, Enza
    Santucci, Maria Alessandra
    BRITISH JOURNAL OF HAEMATOLOGY, 2008, 141 (06) : 899 - 902
  • [27] Identification of signaling pathways involving the CRKL adaptor protein in normal and p210BCR/ABL-transformed hematopoietic cells.
    Uemura, N
    Salgia, R
    Sattler, M
    Okuda, K
    Prasad, K
    Groffen, J
    Griffin, JD
    BLOOD, 1995, 86 (10) : 1040 - 1040
  • [28] TYROSINE PHOSPHORYLATION OF P95(VAV) IN MYELOID CELLS IS REGULATED BY GM-CSF, IL-3 AND STEEL FACTOR AND IS CONSTITUTIVELY INCREASED BY P210(BCR/ABL)
    MATSUGUCHI, T
    INHORN, RC
    CARLESSO, N
    XU, G
    DRUKER, B
    GRIFFIN, JD
    EMBO JOURNAL, 1995, 14 (02): : 257 - 265
  • [29] Inhibition of Bcr-Abl kinase activity by PD180970 blocks constitutive activation of Stat5 and growth of CML cells
    Huang, M
    Dorsey, JF
    Epling-Burnette, PK
    Nimmanapalli, R
    Landowski, TH
    Mora, LB
    Niu, GL
    Sinibaldi, D
    Bai, FQ
    Kraker, A
    Yu, H
    Moscinski, L
    Wei, S
    Djeu, J
    Dalton, WS
    Bhalla, K
    Loughran, TP
    Wu, J
    Jove, R
    ONCOGENE, 2002, 21 (57) : 8804 - 8816
  • [30] P210 Bcr-Abl interacts with the interleukin 3 receptor beta(c) subunit and constitutively induces its tyrosine phosphorylation
    WilsonRawls, J
    Xie, SH
    Liu, JX
    Laneuville, P
    Arlinghaus, RB
    CANCER RESEARCH, 1996, 56 (15) : 3426 - 3430