The Mitochondrial-Derived Peptide MOTS-c Enhances Pancreatic Beta-Cell Function and Survival by Ameliorating Endoplasmic Reticulum Stress

被引:0
|
作者
Min, Se Hee
Shen, Hai Ying
Moon, Sun Joon
Ahn, Chang Ho
Cho, Young Min
机构
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
2172-P
引用
收藏
页码:A569 / A569
页数:1
相关论文
共 50 条
  • [41] Adiponectin treatment improves insulin resistance in mice by regulating the expression of the mitochondrial-derived peptide MOTS-c and its response to exercise via APPL1–SIRT1–PGC-1α
    Qi Guo
    Bo Chang
    Qiong-li Yu
    Si-tong Xu
    Xue-jie Yi
    Shi-cheng Cao
    Diabetologia, 2020, 63 : 2675 - 2688
  • [42] Modulation of Autophagy Influences the Function and Survival of Human Pancreatic Beta Cells Under Endoplasmic Reticulum Stress Conditions and in Type 2 Diabetes
    Bugliani, M.
    Mossuto, S.
    Grano, F.
    Suleiman, M.
    Marselli, L.
    Boggi, U.
    De Simone, P.
    Eizirik, D. L.
    Cnop, M.
    Marchetti, P.
    De Tata, V.
    FRONTIERS IN ENDOCRINOLOGY, 2019, 10
  • [43] The Validation of Home Urinary C-Peptide as a Novel Method for Measuring Beta-Cell Function after Pancreatic Islet Transplantation
    Oram, Richard A.
    Mcdonald, Timothy J.
    Brooks, Augustin M.
    Forbes, Shareen
    Rutter, Martin
    Smith, Richard
    Choudhary, Pratik
    Rosenthal, Miranda
    Johnson, Paul R.
    Shaw, James
    Hattersley, Andrew T.
    DIABETES, 2013, 62 : A35 - A35
  • [44] PANCREATIC BETA-CELL FUNCTION IN CIRRHOTIC-PATIENTS WITH AND WITHOUT OVERT DIABETES - C-PEPTIDE RESPONSE TO GLUCAGON AND TO MEAL
    MARCHESINI, G
    MELLI, A
    CHECCHIA, GA
    MATTIOLI, L
    CAPELLI, M
    CASSARANI, S
    ZOLI, M
    PISI, E
    METABOLISM-CLINICAL AND EXPERIMENTAL, 1985, 34 (08): : 695 - 701
  • [45] Differential expression of glucagon-like peptide-2 (GLP-2) is involved in pancreatic islet cell adaptations to stress and beta-cell survival
    Khan, Dawood
    Vasu, Srividya
    Moffett, R. Charlotte
    Irwin, Nigel
    Flatt, Peter R.
    PEPTIDES, 2017, 95 : 68 - 75
  • [46] Susceptibility of NODk Compared to C56BL/10 Mice to Nonimmune Islet Beta-Cell Failure Due to Greater Susceptibility to Endoplasmic Reticulum Stress
    Delghingaro-Augusto, Viviane
    Socha, Luis A.
    Khan, Ainy
    Chan, Jeng Y.
    Laybutt, D. Ross
    Nolan, Christopher J.
    DIABETES, 2014, 63 : A538 - A538
  • [47] c-Abl Phosphorylates MFN2 to Regulate Mitochondrial Morphology in Cells under Endoplasmic Reticulum and Oxidative Stress, Impacting Cell Survival and Neurodegeneration
    Martinez, Alexis
    Lamaizon, Cristian M.
    Valls, Cristian
    Llambi, Fabien
    Leal, Nancy
    Fitzgerald, Patrick
    Guy, Cliff
    Kaminski, Marcin M.
    Inestrosa, Nibaldo C.
    van Zundert, Brigitte
    Cancino, Gonzalo I.
    Dulcey, Andres E.
    Zanlungo, Silvana
    Marugan, Juan J.
    Hetz, Claudio
    Green, Douglas R.
    Alvarez, Alejandra R.
    ANTIOXIDANTS, 2023, 12 (11)
  • [48] PANCREATIC BETA-CELL FUNCTION EVALUATED BY INTRAVENOUS GLUCOSE AND GLUCAGON STIMULATION - A COMPARISON BETWEEN INSULIN AND C-PEPTIDE TO MEASURE INSULIN-SECRETION
    GOTTSATER, A
    LANDINOLSSON, M
    FERNLUND, P
    GULLBERG, B
    LERNMARK, A
    SUNDKVIST, G
    SCANDINAVIAN JOURNAL OF CLINICAL & LABORATORY INVESTIGATION, 1992, 52 (07): : 631 - 639
  • [49] Two Cases of Mitochondrial Diabetes in Which Pancreatic Beta-Cell Function and Neuropathy Were Improved by Glucagon-Like Peptide-1 Receptor Agonist Therapy
    Minezaki, Midori
    Abe, Ichiro
    Koga, Monami
    Sugimoto, Kaoru
    Takeshita, Kaori
    Takahara, Saori
    Nakagawa, Midori
    Fujii, Hideyuki
    Ohishi, Hanako
    Kudo, Tadachika
    Ohe, Kenji
    Yanase, Toshihiko
    Kobayashi, Kunihisa
    JOURNAL OF ENDOCRINOLOGY AND METABOLISM, 2019, 9 (1-2) : 33 - 36
  • [50] The human glucagon-like peptide-1 analogue liraglutide preserves pancreatic beta cells via regulation of cell kinetics and suppression of oxidative and endoplasmic reticulum stress in a mouse model of diabetes
    Shimoda, M.
    Kanda, Y.
    Hamamoto, S.
    Tawaramoto, K.
    Hashiramoto, M.
    Matsuki, M.
    Kaku, K.
    DIABETOLOGIA, 2011, 54 (05) : 1098 - 1108