Bone marrow stroma (BMS) prevents apoptosis of lymphoma cells by upregulation of BMS-derived B-cell activating factor (BAFF) and activation of NF-kB.

被引：0

作者：

Lwin, Tint ^{[1
]}

Hazlehurst, Lori A. ^{[1
]}

Moscinski, Lynn C. ^{[1
]}

Dalton, William S. ^{[1
]}

Tao, Jianguo ^{[1
]}

机构：

[1] Univ S Florida, H Lee Moffitt Canc Ctr, Dept Interdisciplinary Oncol, Tampa, FL 33682 USA

来源：

BLOOD | 2006年 / 108卷 / 11期

关键词：

D O I：

10.1182/blood.V108.11.2378.2378

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

2378

引用

页码：674A / 674A

页数：1

共 37 条

[31] Constitutive activation of NF-κB causes resistance to apoptosis in human cutaneous T cell lymphoma HuT-78 cells -: Autocrine role of tumor necrosis factor and reactive oxygen intermediates
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[32] Monocyte-lineage cells induced from bone marrow CD34+ progenitor cells of rheumatoid arthritis patients have the capacity to activate IgD+ B cells through direct cellular interactions that do not involve B cell activating factor belonging to the TNF family (BAFF).
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[33] Bone Marrow Mesenchymal Stem Cells (BMSC)-Derived MicroRNA-189 Inhibits Glioma Tumorigenesis Through Suppressing Tumor Necrosis Factor-⟪ (TNF-⟪)-Mediated Nuclear Factor Kappa Light Chain Enhancer of Activated B Cells (NF-KB) Signaling Pathway
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[35] Human bone marrow-derived mesenchymal stem cells promote the growth and drug-resistance of diffuse large B-cell lymphoma by secreting IL-6 and elevating IL-17A levels
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[36] Bispecific antibody fragments with CD20 x CD28 specificity allow effective autologous and allogeneic T-cell activation against malignant cells in peripheral blood and bone marrow cultures from patients with B-cell lineage leukemia and lymphoma
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[37] Platelet-derived growth factor BB mediates the glioma-induced migration of bone marrow-derived mesenchymal stem cells by promoting the expression of vascular cell adhesion molecule-1 through the PI3K, P38 MAPK and NF-κB pathways
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