Exercise Physiology in Heart Failure and Preserved Ejection Fraction

被引：43

作者：

Haykowsky, Mark I. ^{[1
]}

Kitzman, Dalane W. ^{[2
]}

机构：

[1] Univ Alberta, Mazankowski Alberta Heart Inst, Alberta Cardiovasc & Stroke Res Ctr ABACUS, Fac Rehabil Med, Edmonton, AB T6G 2G4, Canada

[2] Wake Forest Sch Med, Dept Internal Med, Cardiol Sect, Winston Salem, NC 27157 USA

来源：

HEART FAILURE CLINICS | 2014年 / 10卷 / 03期

关键词：

Heart failure and preserved ejection fraction; Exercise physiology; Physical conditioning; VENTRICULAR SYSTOLIC FUNCTION; MEDIATED ARTERIAL DILATION; OLDER PATIENTS; SKELETAL-MUSCLE; AEROBIC CAPACITY; ELDERLY-PATIENTS; INTOLERANCE; DETERMINANTS; STIFFNESS;

D O I：

10.1016/j.hfc.2014.04.001

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Recent advances in the pathophysiology of exercise intolerance in patients with heart failure with preserved ejection fraction (HFPEF) suggest that noncardiac peripheral factors contribute to the reduced peak Vo(2) (peak exercise oxygen uptake) and to its improvement after endurance exercise training. A greater understanding of the peripheral skeletal muscle vascular adaptations that occur with physical conditioning may allow for tailored exercise rehabilitation programs. The identification of specific mechanisms that improve whole body and peripheral skeletal muscle oxygen uptake could establish potential therapeutic targets for medical therapies and a means to follow therapeutic response.

引用

页码：445 / +

页数：9

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