Egr-1 abrogates the block imparted by c-Myc on terminal myeloid differentiation without arresting proliferation

被引：0

作者：

Shafarenko, M ^{[1
]}

Liebermann, DA ^{[1
]}

Hoffman, B ^{[1
]}

机构：

[1] Temple Univ, Sch Med, Fels Inst Canc Res & Mol Biol, Philadelphia, PA 19122 USA

来源：

BLOOD | 2002年 / 100卷 / 11期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

4340

引用

页码：208B / 208B

页数：1

共 47 条

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[22] alpha(1)-adrenergic stimulation of isolated rat atria results in discoordinate increases in natriuretic peptide secretion and gene expression and enhances Egr-1 and c-Myc expression
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[23] LncRNA KCNQ1OT1 controls cell proliferation, differentiation and apoptosis by sponging miR-326 to regulate c-Myc expression in acute myeloid leukemia
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[24] Deregulated c-Myc prematurely recruits both Type I and IICD95/Fas apoptotic pathways associated with terminal myeloid differentiation
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[27] The addition of the estrogen receptor to the carboxy terminus of c-Myc inhibits the Fas/CD-95 apoptotic pathway associated with the Myc-mediated block in myeloid differentiation.
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[28] MOLECULAR GENETIC-EVIDENCE FOR A DIFFERENTIATION PROLIFERATION COUPLING DURING DMSO-INDUCED MYELOID MATURATION OF HL-60 CELLS - ROLE OF THE TRANSCRIPTION ELONGATION BLOCK IN THE C-MYC GENE
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[29] Ovol2 Suppresses Cell Cycling and Terminal Differentiation of Keratinocytes by Directly Repressing c-Myc and Notch1
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JOURNAL OF BIOLOGICAL CHEMISTRY, 2009, 284 (42) : 29125 - 29135
[30] Deregulated c-Myc prematurely recruits both Type I and II CD95/Fas apoptotic pathways associated with terminal myeloid differentiation
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Dan A Liebermann
Barbara Hoffman
Oncogene, 2002, 21 : 1600 - 1610

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