Structural recognition of an optimized substrate for the ephrin family of receptor tyrosine kinases

被引:19
|
作者
Davis, Tara L. [1 ,2 ]
Walker, John R. [1 ]
Allali-Hassani, Abdellah [1 ]
Parker, Sirlester A. [3 ]
Turk, Benjamin E. [3 ]
Dhe-Paganon, Sirano [1 ,2 ]
机构
[1] Univ Toronto, Struct Genom Consortium, Toronto, ON M5G 1L7, Canada
[2] Univ Toronto, Dept Physiol, Toronto, ON M5G 1L7, Canada
[3] Yale Univ, Sch Med, Dept Pharmacol, New Haven, CT 06510 USA
基金
美国国家卫生研究院;
关键词
ephrin kinase; peptide array; receptor tyrosine kinase; substrate recognition; X-ray crystallography; MULTIPLE SEQUENCE ALIGNMENT; EPHA3; RECEPTOR; PROTEIN; MUTATIONS; INTERFACE; GENES;
D O I
10.1111/j.1742-4658.2009.07147.x
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Ephrin receptor tyrosine kinase A3 (EphA3, EC 2.7.10.1) is a member of a unique branch of the kinome in which downstream signaling occurs in both ligand- and receptor-expressing cells. Consequently, the ephrins and ephrin receptor tyrosine kinases often mediate processes involving cell-cell contact, including cellular adhesion or repulsion, developmental remodeling and neuronal mapping. The receptor is also frequently overexpressed in invasive cancers, including breast, small-cell lung and gastrointestinal cancers. However, little is known about direct substrates of EphA3 kinase and no chemical probes are available. Using a library approach, we found a short peptide sequence that is a good substrate for EphA3 and is suitable for co-crystallization studies. Complex structures show multiple contacts between kinase and substrates; in particular, two residues undergo conformational changes and by mutation are found to be important for substrate binding and turnover. In addition, a difference in catalytic efficiency between EPH kinase family members is observed. These results provide insight into the mechanism of substrate binding to these developmentally integral enzymes.
引用
收藏
页码:4395 / 4404
页数:10
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