beta-catenin, but not VE-cadherin, colocalizes with the small GTPases rho and RAC at the lamellipodia of migrating endothelial cells.

被引：0
|
作者：
Woolkalis, MJ [1 ]
Ukropec, JA [1 ]
Hollinger, MK [1 ]
机构：
[1] THOMAS JEFFERSON UNIV,DEPT PHYSIOL,PHILADELPHIA,PA 19107
来源：
MOLECULAR BIOLOGY OF THE CELL | 1997年 / 8卷
关键词：
D O I：
暂无
中图分类号：
Q2 [细胞生物学];
学科分类号：
071009 ; 090102 ;
摘要：
引用
收藏
页码：1763 / 1763
页数：1
相关论文
共 27 条
  • [11] The effect of tyrosine kinase inhibitors on the phosphorylation of VE-cadherin in bovine retinal capillary endothelial cells.
    Gillies, MC
    Caruso, L
    INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE, 2000, 41 (04) : S410 - S410
  • [12] Phosphorylation of VE-cadherin controls endothelial phenotypes via p120-catenin coupling and Rac1 activation
    Hatanaka, Kunihiko
    Simons, Michael
    Murakami, Masahiro
    AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2011, 300 (01): : H162 - H172
  • [13] Interaction of VE-cadherin & connexin43 in endothelial cells and its impact on transendothelial migration of tumour cells.
    Cai, J
    Jiang, WG
    Mansel, RE
    BRITISH JOURNAL OF CANCER, 1999, 80 : 37 - 37
  • [14] Fluid shear-induced increases in the tyrosine phosphorylation of catenins associated with VE-cadherin correlate with the loss of SHP2 from VE-cadherin complexes in human endothelial cells.
    Ukropec, JA
    Hollinger, MK
    Woolkalis, MJ
    FASEB JOURNAL, 1999, 13 (04): : A530 - A530
  • [15] eNOS-derived nitric oxide regulates endothelial barrier function through VE-cadherin and Rho GTPases (vol 126 pg 5541, 2013)
    Di Lorenzo, Annarita
    Lin, Michelle I.
    Murata, Takahisa
    Landskroner-Eiger, Shira
    Schleicher, Michael
    Kothiya, Milankumar
    Iwakiri, Yasuko
    Yu, Jun
    Huang, Paul L.
    Sessa, William C.
    JOURNAL OF CELL SCIENCE, 2014, 127 (09) : 2120 - 2120
  • [16] Regulation of VE-cadherin and fli-1 genes by the Ets-1 transcription factor in endothelial cells.
    Lelièvre, E
    Lionneton, F
    Mattot, V
    Spruyt, N
    Stehelin, D
    Soncin, F
    JOURNAL OF MOLECULAR MEDICINE-JMM, 2002, 80 (09): : B6 - B6
  • [17] Inflammation-induced subcellular redistribution of VE-cadherin, actin, and γ-catenin in cultured human lung microvessel endothelial cells
    Lim, MJ
    Chiang, ET
    Hechtman, HB
    Shepro, D
    MICROVASCULAR RESEARCH, 2001, 62 (03) : 366 - 382
  • [18] Inhibition of the expression of VE-cadherin/catenin complex by gamma linolenic acid in human vascular endothelial cells, and its impact on angiogenesis
    Cai, J
    Jiang, WG
    Mansel, RE
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1999, 258 (01) : 113 - 118
  • [19] Vascular hyperpermeability inducer thrombin causes beta-catenin translocation in human umbilical vein endothelial cells.
    Beckers, CML
    Engelse, MA
    Musters, RJP
    van Hinsbergh, VWM
    Amerongen, GPV
    JOURNAL OF VASCULAR RESEARCH, 2005, 42 : 97 - 97
  • [20] Sevoflurane prevents lipopolysaccharide-induced barrier dysfunction in human lung microvascular endothelial cells: Rho-mediated alterations of VE-cadherin
    Huang, Yiran
    Tan, Qindong
    Chen, Rui
    Cao, Biao
    Li, Wenhong
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2015, 468 (1-2) : 119 - 124
123