Renal involvement in primary Sjogren's syndrome

被引：56

作者：

Evans, Rhys ^{[1
]}

Zdebik, Anselm ^{[1
]}

Ciurtin, Coziana ^{[2
]}

Walsh, Stephen B. ^{[1
]}

机构：

[1] UCL, Sch Med, UCL Ctr Nephrol, London NW3 2PF, England

[2] Univ Coll London Hosp, Dept Rheumatol, London, England

来源：

RHEUMATOLOGY | 2015年 / 54卷 / 09期

关键词：

Sjogren's syndrome; tubulointerstitial nephritis; autoimmune epithelialitis; Th17; cells; B cells; distal renal tubular acidosis; Fanconi syndrome; autoantibodies; vasculitis; hypocomplementaemia; CARBONIC-ANHYDRASE-II; TUBULAR-ACIDOSIS; DOUBLE-BLIND; INTERSTITIAL NEPHRITIS; RITUXIMAB TREATMENT; SALIVARY-GLANDS; AUTOIMMUNE; AUTOANTIBODIES; PATIENT; CELLS;

D O I：

10.1093/rheumatology/kev223

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

SS is a prevalent and underdiagnosed systemic disease that primarily affects epithelial tissue. It may affect renal function either as epithelial disease causing tubulointerstitial nephritis or as an immune complex-mediated glomerulopathy. These lesions may cause a variety of clinical features, both overt and occult. The epithelial disease is mediated by B and T cells, notably the Th17 subtype. We review the prevalence of renal SS, its presentation, likely pathogenesis and treatment.

引用

页码：1541 / 1548

页数：8

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