c-Src Kinase Inhibition Reduces Arrhythmia Inducibility and Connexin43 Dysregulation After Myocardial Infarction

被引：0

作者：

Rutledge, Cody A. ^{[1
]}

Ng, Fu S. ^{[2
]}

Sulkin, Matthew S. ^{[2
]}

Greener, Ian D. ^{[1
]}

Sergeyenko, Artem M. ^{[1
]}

Liu, Hong ^{[1
]}

Gemel, Joanna ^{[3
]}

Beyer, Eric C. ^{[3
]}

Sovari, Ali A. ^{[1
]}

Efimov, Igor R. ^{[2
]}

Dudley, Samuel C. ^{[1
]}

机构：

[1] UIC, Chicago, IL USA

[2] Washington Univ, St Louis, MO USA

[3] Univ Chicago, Chicago, IL 60637 USA

来源：

CIRCULATION | 2013年 / 128卷 / 22期

关键词：

Connexin; Myocardial infarction; Sudden cardiac death; Arrhythmias; treatment of;

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

14640

引用

页数：1

共 42 条

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[22] A promising therapy against human glioblastoma stem cells: cell-penetrating peptides based on the interaction between connexin43 and c-Src
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[23] A Peptide Containing the Carboxy-Terminal Domain of Connexin43 Reduces Arrhythmias and Improves Cardiac Function after Myocardial Injury
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[25] Structural changes in the carboxyl terminus of the gap junction protein connexin43 indicates signaling between binding domains for c-Src and zonula occludens-1
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[26] Inhibition of protein kinase c-src reduces the incidence of breast cancer metastases and increases survival in mice: Implications for therapy
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[27] Inhibition of PCSK9 reduces infarct size and arrhythmia susceptibility in cardiac ischeamia/reperfusion injury through attenuating mitochondrial dysfunction and increasing connexin43 phosphorylation
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[28] Inhibition of protein kinase C reduces left ventricular fibrosis and dysfunction following myocardial infarction
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[29] Inhibition of gap junctional intercellular communication by tumor promoters in connexin43 and connexin32-expressing liver cells: Cell specificity and role of protein kinase C
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[30] Structure of the carboxyl terminus of the gap junction protein Connexin43 (Cx43) and changes induced by binding of c-Src SH3 and ZO-1 PDZ-2 domains
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