ATP6AP2, wnt-pathways and the cell cycle

被引：0
|
作者：
Peters, J. [1 ]
机构：
[1] Univ Med Greifswald, Physiol, Greifswald, Germany
来源：
ACTA PHYSIOLOGICA | 2017年 / 219卷
关键词：
D O I：
暂无
中图分类号：
Q4 [生理学];
学科分类号：
071003 ;
摘要：
S09-2
引用
收藏
页码：19 / 19
页数：1
相关论文
共 50 条
  • [31] First case of sporadic ATP6AP2 Mutation reported in Asia in a Parkinson's disease patient
    Shukla, K.
    Sawal, N.
    MOVEMENT DISORDERS, 2023, 38 : S501 - S502
  • [32] Atp6ap2 deletion causes extensive vacuolation that consumes the insulin content of pancreatic beta cells
    Binger, K. J.
    Neukam, M.
    Tattikota, S. Gopal
    Qadri, F.
    Puchkov, D.
    Poy, M.
    Solimena, M.
    Muller, D. N.
    Birkenfeld, A.
    DIABETOLOGIA, 2019, 62 : S230 - S230
  • [33] (Pro)renin receptor/ATP6AP2 is required for autophagy and regulates proliferation in lung adenocarcinoma cells
    Ohba, Koji
    Endo, Moe
    Sato, Shigemitsu
    Kashio-Yokota, Yurina
    Hirose, Takuo
    Takahashi, Kazuhiro
    GENES TO CELLS, 2020, 25 (12) : 782 - 795
  • [34] The Atp6ap2/(Pro)-renin receptor is involved in Receptor Mediated Endocytosis by the Renal Proximal Tubule
    Figueiredo, M.
    Sihn, G.
    Mueller, D.
    Nguyen, G.
    Bader, M.
    Bader, M.
    Wagner, C. A.
    ACTA PHYSIOLOGICA, 2016, 216
  • [35] Characterization of Atp6ap2 ((Pro)-Renin Receptor) in Receptor Mediated Endocytosis by the Renal Proximal Tubule
    Figueiredo, Marta
    Sihn, Gabin
    Mueller, Dominik
    Genevieve Nguyen
    Bader, Michael
    Wagner, Carsten
    FASEB JOURNAL, 2015, 29
  • [36] The (pro)renin receptor/ATP6ap2 is expressed in the murine hippocampus by adult and newly generated neurons
    Schaefer, S. T.
    Peters, J.
    Halbach, O. von Bohlen und
    RESTORATIVE NEUROLOGY AND NEUROSCIENCE, 2013, 31 (02) : 225 - 231
  • [37] ATP6AP2 over-expression causes morphological alterations in the hippocampus and in hippocampus-related behaviour
    Bracke, A.
    Schaefer, S.
    Halbach, V. von Bohlen und
    Klempin, F.
    Bente, K.
    Bracke, K.
    Staar, D.
    van den Brandt, J.
    Harzsch, S.
    Bader, M.
    Wenzel, U. O.
    Peters, J.
    Halbach, O. Von Bohlen Und
    BRAIN STRUCTURE & FUNCTION, 2018, 223 (05): : 2287 - 2302
  • [38] ATP6AP2 is robustly expressed in pancreatic β cells and neuroendocrine tumors, and plays a role in maintaining cellular viability
    Taguchi, Tomomi
    Kimura, Kaori
    Suzuki, Agena
    Fujishima, Rei
    Shimizu, Naoya
    Hoshiyama, Ayako
    Masaki, Tsuguto
    Inoue, Mitsuko
    Kato, Yukiko
    Satomi, Takebe
    Takano, Koji
    Imada, Tasuku
    Sasaki, Shugo
    Miyatsuka, Takeshi
    SCIENTIFIC REPORTS, 2023, 13 (01)
  • [39] ATP6AP2 is robustly expressed in pancreatic β cells and neuroendocrine tumors, and plays a role in maintaining cellular viability
    Tomomi Taguchi
    Kaori Kimura
    Agena Suzuki
    Rei Fujishima
    Naoya Shimizu
    Ayako Hoshiyama
    Tsuguto Masaki
    Mitsuko Inoue
    Yukiko Kato
    Takebe Satomi
    Koji Takano
    Tasuku Imada
    Shugo Sasaki
    Takeshi Miyatsuka
    Scientific Reports, 13
  • [40] A splice site mutation in ATP6AP2 causes X-linked intellectual disability, epilepsy, and parkinsonism
    Gupta, Harsh V.
    Vengoechea, Jaime
    Sahaya, Kinshuk
    Virmani, Tuhin
    PARKINSONISM & RELATED DISORDERS, 2015, 21 (12) : 1473 - 1475
12345