Differential role of Smad2 and Smad3 in mediating TGFβ1 overexpression induced psoriasis-like skin inflammation

被引：0
|
作者：
Li, A. G.
Siddiqui, Y.
Deng, C.
Wang, X.
机构：
[1] Oregon Hlth Sci Univ, Portland, OR 97201 USA
[2] NIDDK, NIH, Bethesda, MD USA
来源：
JOURNAL OF INVESTIGATIVE DERMATOLOGY | 2006年 / 126卷
关键词：
D O I：
暂无
中图分类号：
R75 [皮肤病学与性病学];
学科分类号：
100206 ;
摘要：
560
引用
收藏
页码：94 / 94
页数：1
相关论文
共 50 条
  • [21] Ontogeny and role in liver regeneration of SMAD2 and SMAD3; Essential components in liver formation
    Mishra, L
    Tang, Y
    Monga, SPS
    Weinstein, M
    Yang, X
    Rashid, A
    Diehl, AM
    Deng, CX
    GASTROENTEROLOGY, 1999, 116 (04) : A1248 - A1248
  • [22] Screening SMAD1, SMAD2, SMAD3, and SMAD5 for germline mutations in juvenile polyposis syndrome
    Bevan, S
    Woodford-Richens, K
    Rozen, P
    Eng, C
    Young, J
    Dunlop, M
    Neale, K
    Phillips, R
    Markie, D
    Rodriguez-Bigas, M
    Leggett, B
    Sheridan, E
    Hodgson, S
    Iwama, T
    Eccles, D
    Bodmer, W
    Houlston, R
    Tomlinson, I
    GUT, 1999, 45 (03) : 406 - 408
  • [23] The role of Smad2 and Smad3 in regulating homeostatic functions of fibroblasts in vitro and in adult mice
    Huang, Shuaibo
    Chen, Bijun
    Humeres, Claudio
    Alex, Linda
    Hanna, Anis
    Frangogiannis, Nikolaos G.
    BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH, 2020, 1867 (07):
  • [24] TGF-β1 responsive Smad2 and Smad3 associated proteins identified by tandem affinity purification and mass spectrometry
    Brown, Kimberly A.
    Ham, Amy
    Law, Brian K.
    Chytil, Anna
    Pietenpol, Jennifer A.
    Moses, Harold L.
    CANCER RESEARCH, 2006, 66 (08)
  • [25] TGF-βRⅠ、Smad2、Smad3及Smad7在瘢痕疙瘩中的表达
    孙燚
    宋建星
    汪滋民
    陈江萍
    陈颖
    谭晓洁
    白晋
    中华整形外科杂志, 2006, (05) : 368 - 370
  • [26] Activation of Smad2 but not Smad3 is required to mediate TGF-β signaling during axolotl limb regeneration
    Denis, Jean-Francois
    Sader, Fadi
    Gatien, Samuel
    Villiard, Eric
    Philip, Anie
    Roy, Stephane
    DEVELOPMENT, 2016, 143 (19): : 3481 - 3490
  • [27] SMAD3 and SMAD4 have a more dominant role than SMAD2 in TGFβ-induced chondrogenic differentiation of bone marrow-derived mesenchymal stem cells
    de Kroon, Laurie M. G.
    Narcisi, Roberto
    van den Akker, Guus G. H.
    Vitters, Elly L.
    Davidson, Esmeralda N. Blaney
    van Osch, Gerjo J. V. M.
    van der Kraan, Peter M.
    SCIENTIFIC REPORTS, 2017, 7
  • [28] Smad2Δexon3 and Smad3 have distinct properties in signal transmission leading to TGF-β-induced cell motility
    Yokoyama, Takashi
    Kuga, Takahito
    Itoh, Yuka
    Otake, Shigeo
    Omata, Chiho
    Saitoh, Masao
    Miyazawa, Keiji
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2023, 299 (02)
  • [29] SMAD3 and SMAD4 have a more dominant role than SMAD2 in TGFβ-induced chondrogenic differentiation of bone marrow-derived mesenchymal stem cells
    Laurie M. G. de Kroon
    Roberto Narcisi
    Guus G. H. van den Akker
    Elly L. Vitters
    Esmeralda N. Blaney Davidson
    Gerjo J. V. M. van Osch
    Peter M. van der Kraan
    Scientific Reports, 7
  • [30] Selection of Smad2 for TGF-β to suppress dendritic cells by STAT3/c-Ski-induced repression of Smad3
    Yoon, Jeong-Hwan
    Bae, Eunjin
    Sudo, Katsuko
    Takanashi, Masakatsu
    Han, Jin Soo
    Park, Seok Hee
    Weinstein, Michael
    Nakae, Susumu
    Yamashita, Tadashi
    Lee, In-Kyu
    Ju, Ji Hyeon
    Sumida, Takayuki
    Kuroda, Masahiko
    Miyazawa, Keiji
    Kato, Mitsuyasu
    Mamura, Mizuko
    CANCER RESEARCH, 2017, 77
12345