Viral oncogenesis and its role in nonmelanoma skin cancer

被引：42

作者：

Arron, S. Tuttleton ^{[2
]}

Jennings, L. ^{[1
]}

Nindl, I. ^{[3
]}

Rosl, F. ^{[4
]}

Bavinck, J. N. Bouwes ^{[5
]}

Seckin, D. ^{[6
]}

Trakatelli, M. ^{[7
]}

Murphy, G. M. ^{[1
]}

机构：

[1] Beaumont Hosp, Dept Dermatol, Dublin 9, Ireland

[2] Univ Calif San Francisco, Dept Dermatol, San Francisco, CA 94143 USA

[3] German Canc Res Ctr, Div Viral Skin Carcinogenesis, D-6900 Heidelberg, Germany

[4] German Canc Res Ctr, Div Viral Transformat Mech, D-6900 Heidelberg, Germany

[5] Leiden Univ, Med Ctr, Dept Dermatol, Leiden, Netherlands

[6] Baskent Univ, Fac Med, Dept Dermatol, TR-06490 Ankara, Turkey

[7] Aristotle Univ Thessaloniki, Sch Med, Papageorgiou Hosp, Dept Dermatol, GR-54006 Thessaloniki, Greece

来源：

BRITISH JOURNAL OF DERMATOLOGY | 2011年 / 164卷 / 06期

关键词：

D O I：

10.1111/j.1365-2133.2011.10322.x

中图分类号：

R75 [皮肤病学与性病学];

学科分类号：

100206 ;

摘要：

In recent years, the contribution of viruses to cutaneous oncogenesis has steadily gained recognition. The archetype is human herpesvirus 8, which is well established as the causative agent in Kaposi sarcoma. Other viruses believed to play a role in nonmelanoma skin cancer include human papillomavirus and the recently described Merkel cell polyomavirus. We review the mechanisms by which these three viruses interact with the host cell, ultraviolet radiation and immunosuppression to result in carcinogenesis. © 2011 The Authors. BJD © 2011 British Association of Dermatologists.

引用

页码：1201 / 1213

页数：13

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