Osteoarthritis year 2011 in review: biology

被引：32
|
作者：
Lotz, M. [1 ]
机构：
[1] Scripps Res Inst, Dept Mol & Expt Med, La Jolla, CA 92037 USA
来源：
OSTEOARTHRITIS AND CARTILAGE | 2012年 / 20卷 / 03期
关键词：
Angiogenesis; Autophagy; Lubricin; Mitochondria; GENETICALLY HETEROGENEOUS MICE; EXTENDS LIFE-SPAN; CARTILAGE DEGENERATION; OSTEOCHONDRAL JUNCTION; RHEUMATOID-ARTHRITIS; GROWTH-FACTOR; CELL-DEATH; STEM-CELLS; RAT MODEL; LUBRICIN;
D O I：
10.1016/j.joca.2011.11.015
中图分类号：
R826.8 [整形外科学]; R782.2 [口腔颌面部整形外科学]; R726.2 [小儿整形外科学]; R62 [整形外科学（修复外科学）];
学科分类号：
摘要：
This review is focused on advances in understanding the biology of joint homeostasis and osteoarthritis (OA) pathogenesis mechanisms that have led to proof of concept studies on new therapeutic approaches. The three selected topics include angiogenesis in joint tissues, biomechanics and joint lubrication and mitochondrial dysfunction. This new information represents progress in the integration of mechanisms that control multiple aspects of OA pathophysiology. (C) 2011 Osteoarthritis Research Society International. Published by Elsevier Ltd. All rights reserved.
引用
收藏
页码：192 / 196
页数：5
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