Keratinocyte-specific ablation of the NF-κB regulatory protein A20 (TNFAIP3) reveals a role in the control of epidermal homeostasis

被引：0

作者：

Lippens, Saskia ^{[1
]}

Lefebvre, Sylvie ^{[2
]}

Gilbert, Barbara ^{[1
]}

Sze, Mozes ^{[1
]}

Devos, Michael ^{[1
]}

Verhelst, Kelly ^{[1
]}

Vereecke, Lars ^{[1
]}

McGuire, Conor ^{[1
]}

Guerin, Chris ^{[1
]}

Vandenabeele, Peter ^{[1
]}

Pasparakis, Manolis ^{[3
]}

Mikkola, Marja L. ^{[2
]}

Van Loo, Geert ^{[1
]}

Declercq, Wim ^{[1
]}

机构：

[1] VIB UGhent, Ghent, Belgium

[2] Univ Helsinki, Helsinki, Finland

[3] Univ Cologne, Cologne, Germany

来源：

JOURNAL OF INVESTIGATIVE DERMATOLOGY | 2011年 / 131卷

关键词：

D O I：

暂无

中图分类号：

R75 [皮肤病学与性病学];

学科分类号：

100206 ;

摘要：

引用

页码：S6 / S6

页数：1

共 27 条

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[22] Targeting of TNFAIP3 (A20) Links MiR-125a and MiR-125b to Constitutive Activation of the NF-KB Pathway and Lymphomagenesis
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[23] DNGR1-mediated deletion of A20/Tnfaip3 in dendritic cells alters T and B-cell homeostasis and promotes autoimmune liver pathology
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[26] Intestinal Epithelial-Cell (IEC) Specific Expression of Tumor Necrosis Factor Alpha-Induced Protein 3 (TNFAIP3) Reveals Vital Role for IECs in Mediating Intestinal Inflammation
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