Preserved insulin secretion despite impaired glucose signalling after pancreatic beta cell selective deletion of the tumour suppressor LKB1

被引：0

作者：

Sayers, S. ^{[1
]}

Kantor, C. ^{[1
]}

Pullen, T. J. ^{[1
]}

Nguyen-Tu, M. S. ^{[1
]}

Hodson, D. J. ^{[1
]}

McGinty, J. ^{[2
]}

Lingling, C. ^{[2
]}

French, P. ^{[2
]}

Ibberson, M. ^{[3
]}

Thorens, B. ^{[3
]}

机构：

[1] Univ London Imperial Coll Sci Technol & Med, Sect Cell Biol, Div Diabet Endocrinol & Metab, London, England

[2] Univ London Imperial Coll Sci Technol & Med, Sect Photon, Dept Phys, London, England

[3] Univ Lausanne, Ctr Integrat Genom, Lausanne, Switzerland

来源：

DIABETIC MEDICINE | 2015年 / 32卷

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

A37 (P14)

引用

页码：13 / 13

页数：1

共 38 条

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[35] Wnt4 antagonises Wnt3a mediated increases in growth and glucose stimulated insulin secretion in the pancreatic beta-cell line, INS-1
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[36] CRFR1 and CRFR2 Receptors Are Expressed in Pancreatic Islets, Promote Beta Cell Proliferation and Potentiate Insulin Secretion in a Glucose-Dependent Manner.
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[37] Knocking down Insulin Receptor in Pancreatic Beta Cell lines with Lentiviral-Small Hairpin RNA Reduces Glucose-Stimulated Insulin Secretion via Decreasing the Gene Expression of Insulin, GLUT2 and Pdx1
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Gu, Wenyi
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INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, 2018, 19 (04)
[38] OVEREXPRESSION OF HEXOKINASE-I BUT NOT GLUT1 GLUCOSE-TRANSPORTER ALTERS CONCENTRATION-DEPENDENCE OF GLUCOSE-STIMULATED INSULIN-SECRETION IN PANCREATIC BETA-CELL LINE MIN6
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