Inducible Nitric Oxide Synthase (iNOS) Expression in Cirrhotic Patients with Hepatopulmonary Syndrome

被引：0
|
作者：
Sun, Lifang [1 ]
Zhang, Bingquan [2 ]
Zhang, Na [3 ]
Wang, Yanying [1 ,4 ]
Yu, Jing [2 ]
机构：
[1] Heilongjiang Prov Hosp, Haerbin 150036, Peoples R China
[2] Qiqihaer Med Coll, Affiliated Hosp 2, Qiqihar 16100, Peoples R China
[3] Harbin Univ Commerce, Coll Food Engn, Harbin 150036, Peoples R China
[4] Heilongjiang Prov Hosp, Haerbin 150036, Peoples R China
来源：
ENVIRONMENTAL BIOTECHNOLOGY AND MATERIALS ENGINEERING, PTS 1-3 | 2011年 / 183-185卷
关键词：
inducible nitric oxide synthase; hepatopulmonary syndrome; TNF-ALPHA; OXYGENATION; GLYCINE;
D O I：
10.4028/www.scientific.net/AMR.183-185.2207
中图分类号：
Q81 [生物工程学（生物技术）]; Q93 [微生物学];
学科分类号：
071005 ; 0836 ; 090102 ; 100705 ;
摘要：
The aim of this study was to define that the expression of inducible nitric oxide synthase(iNOS)in cell blocks from ascites was a certain value to evaluate hepatopulmonary syndrome(HPS). iNOS can stimulate the liver and lung to produce excess NO.Excessive production of NO can cause pulmonary vasodilatation and impaire arterial oxygenation. HPS is characterized by shortness of breath and hypoxemia caused by impaired arterial oxygenation due to pulmonary microvascular dilatation in patients with liver disease or PTH. Therefore the positive rates of iNOS was a certain value to evaluate the conditions of HPS.
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收藏
页码：2207 / +
页数：2
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