The Unusual Cross-Reactivity of Anti Muscarinic Receptor 3 Monclonal Antibodies Derived from Salivary Glands of Sjogren's Syndrome Patients to Ro Peptides

被引：0

作者：

Quadri, Syed M. S. ^{[1
]}

Koelsch, Kristi A. ^{[2
]}

Kurien, Biji T. ^{[3
]}

Harris, Valerie M. ^{[4
]}

Scofield, R. Hal ^{[2
]}

机构：

[1] Univ Oklahoma, Hlth Sci Ctr, Med, Oklahoma City, OK USA

[2] US Dept Vet Affairs Med Ctr, Oklahoma City, OK USA

[3] Oklahoma Med Res Fdn, Arthrit & Clin Immunol, 825 NE 13th St, Oklahoma City, OK 73104 USA

[4] Oklahoma Med Res Fdn, 825 NE 13th St, Oklahoma City, OK 73104 USA

来源：

ARTHRITIS & RHEUMATOLOGY | 2016年 / 68卷

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

662

引用

页数：2

共 47 条

[41] Lower frequency of focal lip sialadenitis (focus score) in smoking patients. Can tobacco diminish the salivary gland involvement as judged by histological examination and anti-SSA/Ro and anti-SSB/La antibodies in Sjogren's syndrome?
Manthorpe, R
Benoni, C
Jacobsson, L
Kirtava, Z
Larsson, A
Liedholm, R
Nyhagen, C
Tabery, H
Theander, E
ANNALS OF THE RHEUMATIC DISEASES, 2000, 59 (01) : 54 - 60
[42] Toll-like receptor-3 triggering of salivary gland epithelial cells (SGEC) causes detachment-induced cell death (anoikia) that is more pronounced in cells derived from Sjogren's syndrome patients
Spachidou, M. P.
Kapsogeorgou, E. K.
Moutsopoulos, H. M.
Manoussakis, M. N.
ANNALS OF THE RHEUMATIC DISEASES, 2007, 66 : A36 - A36
[43] Amphipathic variable region heavy chain peptides derived from monoclonal human Wegener's anti-PR3 antibodies stimulate lymphocytes from patients with Wegener's granulomatosis and microscopic polyangiitis
Peen, E
Malone, C
Myers, C
Williams, RC
Peck, AB
Csernok, E
Gross, WL
Staud, R
CLINICAL AND EXPERIMENTAL IMMUNOLOGY, 2001, 125 (02): : 323 - 331
[44] Seroreactivity of the Severe Acute Respiratory Syndrome Coronavirus 2 Recombinant S Protein, Receptor-Binding Domain, and Its Receptor-Binding Motif in COVID-19 Patients and Their Cross-Reactivity With Pre-COVID-19 Samples From Malaria-Endemic Areas
Traore, Abdouramane
Guindo, Merepen A.
Konate, Drissa
Traore, Bourama
Diakite, Seidina A.
Kante, Salimata
Dembele, Assitan
Cisse, Abdourhamane
Incandela, Nathan C.
Kodio, Mamoudou
Coulibaly, Yaya I.
Faye, Ousmane
Kajava, Andrey V.
Pratesi, Federico
Migliorini, Paola
Papini, Anna Maria
Pacini, Lorenzo
Rovero, Paolo
Errante, Fosca
Diakite, Mahamadou
Arevalo-Herrera, Myriam
Herrera, Socrates
Corradin, Giampietro
Balam, Saidou
FRONTIERS IN IMMUNOLOGY, 2022, 13
[45] Neutralizing Anti-Heat-Stable Toxin (STa) Antibodies Derived from Enterotoxigenic Escherichia coli Toxoid Fusions with STa Proteins Containing N12S, L9A/N12S, or N12S/A14T Mutations Show Little Cross-Reactivity with Guanylin or Uroguanylin
Duan, Qiangde
Huang, Jiachen
Xiao, Nan
Seo, Hyesuk
Zhang, Weiping
APPLIED AND ENVIRONMENTAL MICROBIOLOGY, 2018, 84 (02)
[46] GLUTAMATE RECEPTOR ANTIBODIES IN NEUROLOGICAL DISEASES: Anti-AMPA-GluR3 antibodies, Anti-NMDA-NR1 antibodies, Anti-NMDA-NR2A/B antibodies, Anti-mGluR1 antibodies or Anti-mGluR5 antibodies are present in subpopulations of patients with either: Epilepsy, Encephalitis, Cerebellar Ataxia, Systemic Lupus Erythematosus (SLE) and Neuropsychiatric SLE, Sjogren’s syndrome, Schizophrenia, Mania or Stroke. These autoimmune anti-glutamate receptor antibodies can bind neurons in few brain regions, activate glutamate receptors, decrease glutamate receptor’s expression, impair glutamate-induced signaling and function, activate Blood Brain Barrier endothelial cells, kill neurons, damage the brain, induce behavioral/psychiatric/cognitive abnormalities and Ataxia in animal models, and can be removed or silenced in some patients by immunotherapy
Mia Levite
Journal of Neural Transmission, 2014, 121 : 1029 - 1075
[47] GLUTAMATE RECEPTOR ANTIBODIES IN NEUROLOGICAL DISEASES: Anti-AMPA-GluR3 antibodies, Anti-NMDA-NR1 antibodies, Anti-NMDA-NR2A/B antibodies, Anti-mGluR1 antibodies or Anti-mGluR5 antibodies are present in subpopulations of patients with either: Epilepsy, Encephalitis, Cerebellar Ataxia, Systemic Lupus Erythematosus (SLE) and Neuropsychiatric SLE, Sjogren's syndrome, Schizophrenia, Mania or Stroke. These autoimmune anti-glutamate receptor antibodies can bind neurons in few brain regions, activate glutamate receptors, decrease glutamate receptor's expression, impair glutamate-induced signaling and function, activate Blood Brain Barrier endothelial cells, kill neurons, damage the brain, induce behavioral/psychiatric/cognitive abnormalities and Ataxia in animal models, and can be removed or silenced in some patients by immunotherapy
Levite, Mia
JOURNAL OF NEURAL TRANSMISSION, 2014, 121 (08) : 1029 - 1075

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