Transient bone marrow oedema syndrome in an HIV-infected patient on highly active antiretroviral therapy

被引：0

作者：

Vera, J. H. ^{[1
]}

Walker, A. E. ^{[2
]}

Kinderlerer, A. ^{[3
]}

Cooke, G. S. ^{[4
]}

机构：

[1] Univ London Imperial Coll Sci Technol & Med, St Marys Hosp, Dept Infect Dis, London, England

[2] St Marys Hosp, Dept Radiol, London, England

[3] St Marys Hosp, Dept Rheumatol, London, England

[4] Univ KwaZulu Natal, Africa Ctr Hlth & Populat Studies, Imperial Coll London, Dept Infect Dis, Durban, South Africa

来源：

INTERNATIONAL JOURNAL OF STD & AIDS | 2012年 / 23卷 / 03期

关键词：

HIV; bone marrow oedema syndrome; BMES; tenofovir; antiretroviral; REGIONAL MIGRATORY OSTEOPOROSIS; OF-THE-LITERATURE; AVASCULAR NECROSIS; OSTEONECROSIS;

D O I：

10.1258/ijsa.2009.009358

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

Bone marrow oedema syndrome (BMES, also known as transient osteoporosis) is an uncommon, self-limiting condition characterized by disabling pain, reversible osteopaenia on X-rays and by bone marrow oedema pattern on magnetic resonance imaging (MRI). Here we describe the first reported case of BMES in an HIV-positive patient on highly active antiretroviral therapy.

引用

页码：213 / 215

页数：3

共 50 条

[31] Immunological nonresponse to highly active antiretroviral therapy in HIV-infected subjects: Is the bone marrow impairment causing CD4 lymphopenia?
Badolato, Raffaele
[J]. CLINICAL INFECTIOUS DISEASES, 2008, 46 (12) : 1911 - 1912
[32] Prolonged Paradoxical Reaction of Tuberculosis in an HIV-Infected Patient after Initiation of Highly Active Antiretroviral Therapy
A. Ramos
A. Asensio
I. Perales
M. C. Montero
T. Martín
[J]. European Journal of Clinical Microbiology and Infectious Diseases , 2003, 22 : 374 - 376
[33] Regression of Kaposi's sarcoma lesions following highly active antiretroviral therapy in an HIV-infected patient
Kumarasamy, N.
Venkatesh, K. K.
Devaleenol, B.
Poongulali, S.
Ahilasamy, N.
[J]. INTERNATIONAL JOURNAL OF STD & AIDS, 2008, 19 (11) : 786 - 788
[34] Toxoplasma encephalitis in an HIV-infected patient on highly active antiretroviral therapy despite sustained immune response
Atreya, Auras R.
Arora, Sonali
Gadiraju, Vijay T.
Martagon-Villamil, Jose
Skiest, Daniel J.
[J]. INTERNATIONAL JOURNAL OF STD & AIDS, 2014, 25 (05) : 383 - 386
[35] Prolonged paradoxical reaction of tuberculosis in an HIV-infected patient after initiation of highly active antiretroviral therapy
Ramos, A
Asensio, A
Perales, I
Montero, MC
Martín, T
[J]. EUROPEAN JOURNAL OF CLINICAL MICROBIOLOGY & INFECTIOUS DISEASES, 2003, 22 (06) : 374 - 376
[36] Alternative therapy use in HIV-infected patients receiving highly active antiretroviral therapy
Risa, KJ
Nepon, L
Justis, JC
Panwalker, A
Berman, SM
Cinti, S
Wagener, MM
Singh, N
[J]. INTERNATIONAL JOURNAL OF STD & AIDS, 2002, 13 (10) : 706 - 713
[37] Directly observed highly active antiretroviral therapy for HIV-infected children in Cambodia
Myung, Patricia
Pugatch, David
Brady, Mark F.
Many, Phok
Harwell, Joseph I.
Lurie, Mark
Tucker, John
[J]. AMERICAN JOURNAL OF PUBLIC HEALTH, 2007, 97 (06) : 974 - 977
[38] Treatment of tuberculosis in HIV-infected persons in the era of highly active antiretroviral therapy
Dean, GL
Edwards, SG
Ives, NJ
Matthews, G
Fox, EF
Navaratne, L
Fisher, M
Taylor, GP
Miller, R
Taylor, CB
de Ruiter, A
Pozniak, AL
[J]. AIDS, 2002, 16 (01) : 75 - 83
[39] Appendicitis in HIV-infected patients during the era of highly active antiretroviral therapy
Crum-Cianflone, N.
Weekes, J.
Bavaro, M.
[J]. HIV MEDICINE, 2008, 9 (06) : 421 - 426
[40] Arterial stiffness in HIV-infected patients receiving highly active antiretroviral therapy
Sevastianova, K
Sutinen, J
Westerbacka, J
Ristola, M
Yki-Järvinen, H
[J]. ANTIVIRAL THERAPY, 2005, 10 (08) : 925 - 935

← 1 2 3 4 5 →