INTERFERON (IFN) ALPHA PREPARATIONS ACTIVATE HUMAN LUNG MICROVASCULAR ENDOTHELIAL CELLS TO RELEASE THE CHEMOKINE IP10

被引：0

作者：

Mitchell, Jane A. ^{[1
]}

Badiger, Rekha ^{[1
]}

Tietz, Andreas ^{[2
]}

Atkin, Catherine ^{[1
]}

Gashaw, Hime ^{[1
]}

Hinton, Heather J. ^{[2
]}

Hansel, Trevor T. ^{[3
]}

Singer, Thomas ^{[2
]}

Manigold, Tobias ^{[2
]}

机构：

[1] Univ London Imperial Coll Sci Technol & Med, Natl Heart & Lung Inst, London, England

[2] Roche, Basel, Switzerland

[3] St Marys Hosp, Imperial Clin Resp Res Unit ICRRU, London, England

来源：

HEPATOLOGY | 2010年 / 52卷 / 04期

关键词：

D O I：

暂无

中图分类号：

R57 [消化系及腹部疾病];

学科分类号：

摘要：

1012

引用

页码：809A / 810A

页数：2

共 39 条

[31] Enhanced Natural Killer (NK) Cell Recruiting Chemokine Expression in Human Decidual Cells by Tumor Necrosis Factor-alpha (TNF) and Interferon-Gamma (IFN) Affects the Immune Cell Population at the Implantation Site
Lockwood, Charles J.
Smedts, Dineke
Koopman, Louise A.
Buchwalder, Lynn F.
Huang, Joseph S.
Schatz, Frederick
REPRODUCTIVE SCIENCES, 2010, 17 (03) : 151A - 151A
[32] Human rhinovirus induces robust IP-10 release by monocytic cells, which is independent of viral replication but linked to type I interferon receptor ligation and STAT1 activation
Korpi-Steiner, Nichole L.
Bates, Mary Ellen
Lee, Wai-Ming
Hall, David J.
Bertics, Paul J.
JOURNAL OF LEUKOCYTE BIOLOGY, 2006, 80 (06) : 1364 - 1374
[33] Sequential Priming of Human Decidual Cells with a TLR-7 and TLR-9 Agonist Suppresses TNF-α and IFN-γ Enhanced Expression of IP-10, a Natural Killer Cell Recruiting Chemokine.
Faramarzi, S.
Shapiro, J.
Kayisli, U. A.
Schatz, F.
Lockwood, C. J.
REPRODUCTIVE SCIENCES, 2014, 21 (03) : 141A - 141A
[34] The reciprocal regulation of TARC (thymus and activation-regulated chemokine/CCL17) and IP-10 (interferon-induced protein of 10 kDa) by IL-4 but not IL-13 in human keratinocyte cell line, HaCaT cells
Kakinuma, T
Nakamura, K
Wakugawa, M
Yano, S
Saeki, H
Torii, H
Komine, M
Asahina, A
Tamaki, K
JOURNAL OF INVESTIGATIVE DERMATOLOGY, 2002, 119 (01) : 304 - 304
[35] CYTOTOXICITY TRIGGERING FACTOR (CTF) - A NOVEL T-CELL PRODUCT WHICH TRIGGERS TUMOR-NECROSIS-FACTOR-ALPHA (TNF-ALPHA RELEASE BY INTERFERON-GAMMA (IFN-GAMMA) PRIMED HUMAN MONONUCLEAR-CELLS
KORNBLUTH, RS
GREGORY, SA
EDGINGTON, TS
LYMPHOKINE RESEARCH, 1987, 6 (01): : U128 - U128
[36] Peroxisome proliferator-activated receptor-γ activators inhibit IFN-γ-induced expression of the T cell-active CXC chemokines IP-10, Mig, and I-TAC in human endothelial cells
Marx, N
Mach, F
Sauty, A
Leung, JH
Sarafi, MN
Ransohoff, RM
Libby, P
Plutzky, J
Luster, AD
JOURNAL OF IMMUNOLOGY, 2000, 164 (12): : 6503 - 6508
[37] Human Immunodeficiency Virus-1 (hiv) Envelope Protein Gp120-Induced Apoptosis In Lung Microvascular Endothelial Cells Is Mediated Via Concerted Upregulation Of The Chemokine Receptor Cxcr3 And Its Ligand Endothelail Monocyte Attracting Protein Ii (emapii)
Green, L. A.
Yi, R.
Wang, T.
Clauss, M.
AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2013, 187
[38] 13-cis-retinoic acid (CRA) and interferon-alpha (IFN) combination with or without radiation more selectively down regulates BCL-2 and increases apoptosis in prostate tumor cells (LNCAP) than in either human colorectal fibroblast cells (CO-18) or lung fibroblast cells (IMR-91)
Kumar, P
Hill, C
RADIOTHERAPY AND ONCOLOGY, 2004, 73 : S373 - S373
[39] 13-cis-retinoic acid (CRA) and interferon-alpha (IFN) combination with or without radiation has a greater in vitro biological effect in reducing bcl-2 expression and inducing apoptosis in prostate cancer cells (LNCaP) than in normal human colorectal (Co-18) and normal human lung fibrolast (IMR-91) cells: Rationale for a clinical trial
Kumar, P.
Hill, C.
JOURNAL OF CLINICAL ONCOLOGY, 2007, 25 (18)

← 1 2 3 4 →