Roles of Kupffer cells in alcoholic liver disease

被引：9
|
作者：
Takei, Y
Arteel, GE
Bergheim, I
Lambert, JC
McMullen, MR
Nagy, LE
Enomoto, N
Sato, N
机构：
[1] Juntendo Univ, Sch Med, Dept Gastroenterol, Bunkyo Ku, Tokyo 1138421, Japan
[2] Univ Louisville, Ctr Hlth Sci, James Graham Brown Canc Ctr, Dept Pharmacol & Toxicol, Louisville, KY 40292 USA
[3] Case Western Reserve Univ, Dept Nutr, Cleveland, OH 44106 USA
来源：
ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH | 2005年 / 29卷 / 06期
关键词：
Kupffer cells; reactive oxygen species; tumor necrosis factor-alpha; sex difference; thalidomide; PPAR-gamma agonist; metformin;
D O I：
10.1097/01.ALC.0000168169.20591.64
中图分类号：
R194 [卫生标准、卫生检查、医药管理];
学科分类号：
摘要：
This article summarizes presentations at the 2004 International Society for Biomedical Research on Alcoholism held in Heidelberg/Mannheim, Germany. The organizers and chairpersons were Yoshiyuki Takei and Gavin E Arteel. The presentations were on 1) Kupffer cell-derived mediators involved in alcoholic liver disease: reactive oxygen and nitrogen species, by Gavin E. Arteel; 2) Kupffer cell-derived mediators in alcoholic liver disease: tumor necrosis factor-a, by Laura E. Nagy; 3) metformin prevents acute alcohol-induced fat accumulation in mouse liver, by Ina Bergheim; 4) gender difference in alcoholic liver disease, by Nobuyuki Enomoto; and 5) Kupffer cells as a therapeutic target of alcoholic liver disease, by Yoshiyuki Takei.
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页码：1116 / 1120
页数：7
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