Contrasting mechanisms underlie the development of pressure overload versus angiotensin II-induced hypertrophy in mice lacking the NADPH oxidase subunit gp91phox

被引:0
|
作者
Byrne, JA [1 ]
Grieve, DJ [1 ]
Bendall, JK [1 ]
Cave, AC [1 ]
Shah, AM [1 ]
机构
[1] GKT Sch Med, Dept Cardiol, London, England
来源
关键词
D O I
暂无
中图分类号
Q189 [神经科学];
学科分类号
071006 ;
摘要
引用
收藏
页码:42P / 42P
页数:1
相关论文
共 36 条
  • [21] A gp91phox-containing NADPH oxidase contributes to the contractile dysfunction associated with pressure-overload cardiac hypertrophy
    Grieve, DJ
    Byrne, JA
    Layland, J
    Cave, AC
    Shah, AM
    [J]. HEART, 2004, 90 (12)
  • [22] Angiotensin II-dependent chronic hypertension and cardiac hypertrophy are unaffected by gp91phox-containing NADPH oxidase
    Touyz, RM
    Mercure, C
    He, Y
    Javeshghani, D
    Yao, GY
    Callera, GE
    Yogi, A
    Lochard, N
    Reudelhuber, TL
    [J]. HYPERTENSION, 2005, 45 (04) : 530 - 537
  • [23] Reduced renal responses to nitric oxide synthase inhibition in mice lacking the gene for gp91phox subunit of NAD(P)H oxidase
    Haque, Mohammed Z.
    Majid, Dewan S. A.
    [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2008, 295 (03) : F758 - F764
  • [24] Mice lacking the Cβ subunit of PKA are resistant to angiotensin II-induced cardiac hypertrophy and dysfunction
    Enns L.C.
    Bible K.L.
    Emond M.J.
    Ladiges W.C.
    [J]. BMC Research Notes, 3 (1)
  • [25] A gp91phox-containing NAD(P)H Oxidase is a kev mediator of Angiotensin II (AngII)-induced cardiomyocyte hypertrophy
    Hingtgen, SD
    Tian, X
    Sharma, RV
    Davisson, RL
    [J]. FASEB JOURNAL, 2004, 18 (04): : A279 - A280
  • [26] Angiotensin II is not involved in cardiac hypertrophy induced by acute pressure overload in mice lacking angiotensin II type 1a receptor
    Kudoh, S
    Kijima, K
    Matsubara, H
    Sugaya, K
    Murakami, K
    Komuro, I
    [J]. CIRCULATION, 1996, 94 (08) : 3866 - 3866
  • [27] High salt intake delays the hypertensive response to chronic angiotensin II in mice lacking gp91<phox> subunit of NAD(P)H oxidase
    Haque, MZ
    Nomanbhoy, F
    Castillo, A
    Majid, DSA
    [J]. FASEB JOURNAL, 2004, 18 (05): : A1038 - A1038
  • [28] Selective Inhibition of Myocardial NADPH Oxidase with a Dominant-Negative Mutant of p67phox Attenuates Angiotensin II-Induced Cardiac Hypertrophy in Adult Transgenic Mice
    Zhao, Qiong
    Zhao, Guiqing
    Zhang, Chongxu
    Xu, Xianyao
    Varadarajan, Sudhahar
    Urao, Norifumi
    Fukai, Masuko
    Fukai, Tohru
    Xiao, Lei
    [J]. CIRCULATION, 2011, 124 (21)
  • [29] gp91phox is the predominant Nox homologue expressed in cardiomyocytes and siRNA-mediated silencing of its expression abolishes Ang II-induced superoxide generation and cardiomyocyte hypertrophy
    Hingtgen, SD
    Tian, X
    Li, ZB
    Kutschke, W
    Sharma, RV
    Davisson, RL
    [J]. FASEB JOURNAL, 2005, 19 (04): : A701 - A701
  • [30] Myocardial-Specific Overexpression of a Dominant-Negative Mutant of NADPH Oxidase Subunit p67phox Attenuates Pressure-Overload-Induced Cardiac Hypertrophy in Adult Transgenic Mice
    Zhao, Qiong
    Xu, Xianyao
    Zhao, Guiqing
    Zhang, Chongxu
    Deng, Jing
    Christman, John W.
    Xiao, Lei
    [J]. CIRCULATION, 2011, 124 (21)