Molecular mechanisms of MHC class II inhibition by the MARCH1 ubiquitin ligase in response to IL-10

被引：0

作者：

Bourgeois-Daigneault, Marie-Claude ^{[1
]}

Huppe, Gabrielle ^{[2
]}

Tremblay, Jessy ^{[1
]}

Steimle, Viktor ^{[2
]}

Thibodeau, Jacques ^{[1
]}

机构：

[1] Univ Montreal, Dept Microbiol & Immunol, Montreal, PQ H3C 3J7, Canada

[2] Univ Sherbrooke, Fac Sci, Dept Biol, Sherbrooke, PQ J1K 2R1, Canada

来源：

CYTOKINE | 2008年 / 43卷 / 03期

关键词：

D O I：

10.1016/j.cyto.2008.07.186

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：269 / 269

页数：1

共 39 条

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[25] Regulatory Dendritic Cells Restrain NK Cell IFN-γ Production through Mechanisms Involving NKp46, IL-10, and MHC Class I-Specific Inhibitory Receptors
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[26] Mechanisms of the systemic inflammatory response and cardiopulmonary bypass:: A possible role for IL-10 and TGFβ1 in decreased expression of endothelial ICAM-1 and VCAM-1.
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[27] CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10-driven MARCH1-mediated ubiquitination and degradation
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[28] IL-10 indirectly modulates functional activity of CD4+CD28null T-lymphocytes through LFA-3 and HLA class II inhibition
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[29] A synthetic peptide homologous to functional domain of human IL-10 down-regulates expression of MHC class I and transporter associated with antigen processing 1/2 in human melanoma cells
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