C-JUN acts as coactivator or corepressor of inflammatory genes in the JNK signalling patwhay by affecting histone acetylation

被引:0
|
作者
Wolter, S
Wagner, EF
Dittrich-Breiholz, O
Schneider, H
Resech, K
Kracht, M
机构
[1] Med Sch Hannover, Inst Pharmacol, D-30625 Hannover, Germany
[2] Res Inst Mol Pathol, A-1030 Vienna, Austria
关键词
D O I
暂无
中图分类号
R9 [药学];
学科分类号
1007 ;
摘要
265
引用
收藏
页码:74 / 74
页数:1
相关论文
共 50 条
  • [31] Differentiation-associated genes regulated by TPA-induced c-Jun expression via a PKC/JNK pathway in KYSE450 cells
    Yu, XF
    Luo, AP
    Zhou, CC
    Ding, F
    Wu, M
    Zhan, QM
    Liu, ZH
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2006, 342 (01) : 286 - 292
  • [32] HIPK3 Mediates Inflammatory Cytokines and Oxidative Stress Markers in Monocytes in a Rat Model of Sepsis Through the JNK/c-Jun Signaling Pathway
    Liu, Ben
    Hou, Qiuyue
    Ma, Yuhong
    Han, Xuehua
    INFLAMMATION, 2020, 43 (03) : 1127 - 1142
  • [33] HIPK3 Mediates Inflammatory Cytokines and Oxidative Stress Markers in Monocytes in a Rat Model of Sepsis Through the JNK/c-Jun Signaling Pathway
    Ben Liu
    Qiuyue Hou
    Yuhong Ma
    Xuehua Han
    Inflammation, 2020, 43 : 1127 - 1142
  • [34] NEITHER ERK NOR JNK/SAPK MAP KINASE SUBTYPES ARE ESSENTIAL FOR HISTONE H3/HMG-14 PHOSPHORYLATION OR C-FOS AND C-JUN INDUCTION
    CANO, E
    HAZZALIN, CA
    KARDALINOU, E
    BUCKLE, RS
    MAHADEVAN, LC
    JOURNAL OF CELL SCIENCE, 1995, 108 : 3599 - 3609
  • [35] Sophora subprosrate polysaccharide suppress the inflammatory reaction of RAW264.7 cells infected with PCV2 via regulation NF-κB/MAPKs/c-Jun signal pathway and histone acetylation modification
    Yang, Jian
    Cao, Mi-xia
    Hu, Wen-yue
    Wei, Ying-yi
    Hu, Ting-jun
    INTERNATIONAL JOURNAL OF BIOLOGICAL MACROMOLECULES, 2020, 159 : 957 - 965
  • [36] Stimulation of Fas/FasL-mediated apoptosis by luteolin through enhancement of histone H3 acetylation and c-Jun activation in HL-60 leukemia cells
    Wang, Shih-Wei
    Chen, Yun-Ru
    Chow, Jyh-Ming
    Chien, Ming-Hsien
    Yang, Shun-Fa
    Wen, Yu-Ching
    Lee, Wei-Jiunn
    Tseng, Tsui-Hwa
    MOLECULAR CARCINOGENESIS, 2018, 57 (07) : 866 - 877
  • [37] Trichostatin A activates immediate c-jun gene expression by a mechanism involving MEK/MAPK signaling and increased AP-1 activity that is separate from histone acetylation.
    Taher, MM
    Bullock, SK
    Paik, DS
    Hershey, CM
    Allis, DC
    Valerie, K
    CLINICAL CANCER RESEARCH, 1999, 5 : 3777S - 3777S
  • [38] The c-Jun N-terminal Kinase (JNK)-binding Protein (JNKBP1) Acts as a Negative Regulator of NOD2 Protein Signaling by Inhibiting Its Oligomerization Process
    Lecat, Aurore
    Di Valentin, Emmanuel
    Somja, Joan
    Jourdan, Samuel
    Fillet, Marianne
    Kufer, Thomas A.
    Habraken, Yvette
    Sadzot, Catherine
    Louis, Edouard
    Delvenne, Philippe
    Piette, Jacques
    Legrand-Poels, Sylvie
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2012, 287 (35) : 29213 - 29226
  • [39] The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition
    Gao, Yong-Jing
    Xu, Zhen-Zhong
    Liu, Yen-Chin
    Wen, Yeong-Ray
    Decosterd, Isabelle
    Ji, Ru-Rong
    PAIN, 2010, 148 (02) : 309 - 319
  • [40] RETRACTION: Withdrawal: Histone deacetylase inhibitors suppress the induction of c-Jun and its target genes including COX-2 (Retraction of Vol 280, Pg 32569, 2005)
    Yamaguchi, Kentaro
    Lantowski, Agnieszka
    Dannenberg, Andrew J.
    Subbaramaiah, Kotha
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2020, 295 (01) : 294 - 294