Subtyping Pancreatic Cancer

被引：35
|
作者：
Biankin, Andrew V. [1 ,2 ,3 ,4 ]
Maitra, Anirban [5 ]
机构：
[1] Univ Glasgow, Wolfson Wohl Canc Res Ctr, Inst Canc Sci, Glasgow G61 1BD, Lanark, Scotland
[2] Glasgow Royal Infirm, West Scotland Pancreat Unit, Glasgow G31 2ER, Lanark, Scotland
[3] Garvan Inst Med Res, Kinghorn Canc Ctr, Canc Div, Sydney, NSW 2010, Australia
[4] Univ NSW, Fac Med, South Western Sydney Clin Sch, Liverpool, NSW 2170, Australia
[5] Univ Texas MD Anderson Canc Ctr, Sheikh Ahmed Ctr Pancreat Canc Res, Pathol & Translat Mol Pathol, Houston, TX 77030 USA
来源：
CANCER CELL | 2015年 / 28卷 / 04期
基金：
英国惠康基金;
关键词：
THERAPY;
D O I：
10.1016/j.ccell.2015.09.020
中图分类号：
R73 [肿瘤学];
学科分类号：
100214 ;
摘要：
The complex tumor microenvironment can make molecularly subtyping cancer using mRNA expression challenging, particularly for cancers with low epithelial content, such as pancreatic cancer. In a recent edition of Nature Genetics, Moffitt and colleagues show that subtracting normal epithelial transcripts can provide insights into the molecular pathology of pancreatic cancer.
引用
收藏
页码：411 / 413
页数：4
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