Gut-Brain Communication in the Regulation of System Metabolism

被引：0
|
作者：
Mueller, Limo D.
Pfluger, Paul T.
Tschoep, Matthias H. [1 ]
机构：
[1] Tech Univ Munich, IDO, Helmholtz Ctr Munich, Dept Med, Parkring 13, DE-85748 Garching, Germany
来源：
INNATE IMMUNITY: RESISTANCE AND DISEASE-PROMOTING PRINCIPLES | 2013年 / 4卷
关键词：
GLUCAGON-LIKE PEPTIDE-1; RECEPTOR KNOCKOUT MICE; SPONTANEOUS MEAL SIZE; DECREASES FOOD-INTAKE; DIET-INDUCED OBESITY; BODY-WEIGHT; LEPTIN RESPONSIVENESS; INTRAPERITONEAL INJECTION; GENE-EXPRESSION; ENERGY-BALANCE;
D O I：
10.1159/000346539
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
Overweight and obesity are major health threats in modern societies. The regulation of energy metabolism, and thus also hunger and satiety, is a complex process that depends on a constant cross talk of peripheral and central signal mechanisms. To sustain a stable body weight over time, this network must not only take into account the fuel signals from digestive organs but also long-term signals about the energy stored as fat in the body. The aim of this chapter is to summarize the current knowledge of how the gut-brain axis regulates system metabolism. Copyright (C) 2013 S. Karger AG, Basel
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收藏
页码：96 / +
页数：3
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